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Temporal-spatial expression and transcriptional regulation of alpha7 nicotinic acetylcholine receptor by thyroid transcription factor-1 and early growth response factor-1 during murine lung developmentPlausible Roles for RAGE in Conditions Exacerbated by Direct and Indirect (Secondhand) Smoke Exposure.Low anticoagulant heparin targets multiple sites of inflammation, suppresses heparin-induced thrombocytopenia, and inhibits interaction of RAGE with its ligands.High-Mobility Group Box 1 Disrupts Metabolic Function with Cigarette Smoke Exposure in a Ceramide-Dependent Manner.Antenatal exposure of maternal secondhand smoke (SHS) increases fetal lung expression of RAGE and induces RAGE-mediated pulmonary inflammation.Cigarette smoke increases cardiomyocyte ceramide accumulation and inhibits mitochondrial respiration.Immunohistochemical detection and regulation of α5 nicotinic acetylcholine receptor (nAChR) subunits by FoxA2 during mouse lung organogenesisFreezing/Thawing without Cryoprotectant Damages Native but not Decellularized Porcine Renal Tissue.Hypoxia reduces placental mTOR activation in a hypoxia-induced model of intrauterine growth restriction (IUGR).Cigarette smoke extract (CSE) induces RAGE-mediated inflammation in the Ca9-22 gingival carcinoma epithelial cell line.Differential Receptor for Advanced Glycation End Products Expression in Preeclamptic, Intrauterine Growth Restricted, and Gestational Diabetic Placentas.Comparison of four decontamination treatments on porcine renal decellularized extracellular matrix structure, composition, and support of human renal cortical tubular epithelium cells.Malocclusion model of temporomandibular joint osteoarthritis in mice with and without receptor for advanced glycation end products.Automation of Pressure Control Improves Whole Porcine Heart Decellularization.Oral Gingival Cell Cigarette Smoke Exposure Induces Muscle Cell Metabolic DisruptionPrimary alveolar macrophages exposed to diesel particulate matter increase RAGE expression and activate RAGE signaling.Caspase dependent and independent mechanisms of apoptosis across gestation in a sheep model of placental insufficiency and intrauterine growth restriction.Cigarette smoke and decreased oxygen tension inhibit pulmonary claudin-6 expression.Up-Regulation of Claudin-6 in the Distal Lung Impacts Secondhand Smoke-Induced InflammationReceptor for advanced glycation end-products signals through Ras during tobacco smoke-induced pulmonary inflammation.RAGE: developmental expression and positive feedback regulation by Egr-1 during cigarette smoke exposure in pulmonary epithelial cells.Midkine is regulated by hypoxia and causes pulmonary vascular remodeling.Conditional pulmonary over-expression of Claudin 6 during embryogenesis delays lung morphogenesis.Targeted Mutation of Nuclear Bone Morphogenetic Protein 2 Impairs Secondary Immune Response in a Mouse Model.Conditional over-expression of RAGE by embryonic alveolar epithelium compromises the respiratory membrane and impairs endothelial cell differentiation.Cigarette smoke-induced Egr-1 upregulates proinflammatory cytokines in pulmonary epithelial cells.Conditionally induced RAGE expression by proximal airway epithelial cells in transgenic mice causes lung inflammation.LPS from P. gingivalis Negatively Alters Gingival Cell Mitochondrial Bioenergetics.The role of TGF-ß1 in osteoarthritis of the temporomandibular joint in two genetic mouse models.Inhibition of the receptor for advanced glycation end-products (RAGE) protects from secondhand smoke (SHS)-induced intrauterine growth restriction IUGR in mice.Developmental lung expression and transcriptional regulation of claudin-6 by TTF-1, Gata-6, and FoxA2.Osteoarthritis in temporomandibular joint of Col2a1 mutant mice.Combination therapy with folic acid and methionine in the prevention of retinoic acid-induced cleft palate in mice.Differential effect of estrogen receptor alpha and beta agonists on the receptor for advanced glycation end product expression in human microvascular endothelial cells.Decreased activation of placental mTOR family members is associated with the induction of intrauterine growth restriction by secondhand smoke in the mouse.The impact of decellularization agents on renal tissue extracellular matrix.Cardiomyocyte mitochondrial respiration is reduced by receptor for advanced glycation end-product signaling in a ceramide-dependent manner.Ceramides mediate cigarette smoke-induced metabolic disruption in mice.Acute secondhand smoke-induced pulmonary inflammation is diminished in RAGE knockout mice.Efficient decellularization of whole porcine kidneys improves reseeded cell behavior.
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P50
description
researcher ORCID ID = 0000-0002-0931-3025
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name
Paul R Reynolds
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Paul R Reynolds
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Paul R Reynolds
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Paul R Reynolds
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Paul R Reynolds
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Paul R Reynolds
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Paul R Reynolds
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Paul R Reynolds
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Paul R Reynolds
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Paul R Reynolds
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Paul R Reynolds
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Paul R Reynolds
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P1153
56701050600
P31
P496
0000-0002-0931-3025