Axon loss in the spinal cord determines permanent neurological disability in an animal model of multiple sclerosis.
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Virus-mediated autoimmunity in Multiple Sclerosis.Multiple sclerosis: molecular mechanisms and therapeutic opportunitiesNeuroprotective effects of estrogens and androgens in CNS inflammation and neurodegenerationAntibody-Mediated Oligodendrocyte Remyelination Promotes Axon Health in Progressive Demyelinating DiseaseClinico-pathological evidence that axonal loss underlies disability in progressive multiple sclerosisChronic mild stress eliminates the neuroprotective effect of Copaxone after CNS injury.Models of autoimmune demyelination in the central nervous system: on the way to translational medicine.Bringing CLARITY to gray matter atrophy.Laquinimod (ABR-215062) for the treatment of relapsing multiple sclerosis.[Choice of early and escalation treatment options for multiple sclerosis].Axial diffusivity is the primary correlate of axonal injury in the experimental autoimmune encephalomyelitis spinal cord: a quantitative pixelwise analysisReview: Mitochondria and disease progression in multiple sclerosisInitiation and progression of axonopathy in experimental autoimmune encephalomyelitis.Sex differences in autoimmune diseases.Hippocampal CA1 atrophy and synaptic loss during experimental autoimmune encephalomyelitis, EAE.Additive effects of combination treatment with anti-inflammatory and neuroprotective agents in experimental autoimmune encephalomyelitis.Fumaric acid esters exert neuroprotective effects in neuroinflammation via activation of the Nrf2 antioxidant pathway.Contribution of pannexin1 to experimental autoimmune encephalomyelitis.Transgenic inhibition of astroglial NF-kappa B improves functional outcome in experimental autoimmune encephalomyelitis by suppressing chronic central nervous system inflammationNeuroprotection mediated through estrogen receptor-alpha in astrocytesLoss of the receptor tyrosine kinase Axl leads to enhanced inflammation in the CNS and delayed removal of myelin debris during experimental autoimmune encephalomyelitisNeurotrophic cross-talk between the nervous and immune systems: implications for neurological diseases.Targeting experimental autoimmune encephalomyelitis lesions to a predetermined axonal tract system allows for refined behavioral testing in an animal model of multiple sclerosis.Gene therapy in autoimmune, demyelinating disease of the central nervous system.Astrocyte CCL2 sustains immune cell infiltration in chronic experimental autoimmune encephalomyelitis.Pharmaceutical integrated stress response enhancement protects oligodendrocytes and provides a potential multiple sclerosis therapeuticGenetic programs and responses of neural stem/progenitor cells during demyelination: potential insights into repair mechanisms in multiple sclerosis.Estrogen receptor-β ligand treatment after disease onset is neuroprotective in the multiple sclerosis model.Cell therapy for multiple sclerosis.Transfer of myelin-reactive th17 cells impairs endogenous remyelination in the central nervous system of cuprizone-fed mice.Myelin-associated glycoprotein and complementary axonal ligands, gangliosides, mediate axon stability in the CNS and PNS: neuropathology and behavioral deficits in single- and double-null mice.Genetic inactivation of the p66 isoform of ShcA is neuroprotective in a murine model of multiple sclerosis.Preferential Recruitment of Neutrophils into the Cerebellum and Brainstem Contributes to the Atypical Experimental Autoimmune Encephalomyelitis Phenotype.SIRT1 activation confers neuroprotection in experimental optic neuritisAxon loss is an important determinant of weakness in multifocal motor neuropathy.Hyaluronan anchored to activated CD44 on central nervous system vascular endothelial cells promotes lymphocyte extravasation in experimental autoimmune encephalomyelitis.Evaluation of corticospinal axon loss by fluorescent dye tracing in mice with experimental autoimmune encephalomyelitisReversal of axonal loss and disability in a mouse model of progressive multiple sclerosis.Brain regeneration in physiology and pathology: the immune signature driving therapeutic plasticity of neural stem cellsCalpain inhibitor attenuated optic nerve damage in acute optic neuritis in rats.
P2860
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P2860
Axon loss in the spinal cord determines permanent neurological disability in an animal model of multiple sclerosis.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Axon loss in the spinal cord d ...... l model of multiple sclerosis.
@en
Axon loss in the spinal cord d ...... l model of multiple sclerosis.
@nl
type
label
Axon loss in the spinal cord d ...... l model of multiple sclerosis.
@en
Axon loss in the spinal cord d ...... l model of multiple sclerosis.
@nl
prefLabel
Axon loss in the spinal cord d ...... l model of multiple sclerosis.
@en
Axon loss in the spinal cord d ...... l model of multiple sclerosis.
@nl
P2093
P356
P1476
Axon loss in the spinal cord d ...... l model of multiple sclerosis.
@en
P2093
Bruce D Trapp
Carl Bjartmar
Edward Richer
Jerome R Wujek
Richard M Ransohoff
Vincent K Tuohy
P356
10.1093/JNEN/61.1.23
P577
2002-01-01T00:00:00Z