Abnormal cardiac Na(+) channel properties and QT heart rate adaptation in neonatal ankyrin(B) knockout mice.
about
A cardiac arrhythmia syndrome caused by loss of ankyrin-B functionFunctional interaction with filamin A and intracellular Ca2+ enhance the surface membrane expression of a small-conductance Ca2+-activated K+ (SK2) channelSodium channel beta subunits: anything but auxiliaryPost-translational modifications of the cardiac Na channel: contribution of CaMKII-dependent phosphorylation to acquired arrhythmiasMouse models of long QT syndrome.Variable Na(v)1.5 protein expression from the wild-type allele correlates with the penetrance of cardiac conduction disease in the Scn5a(+/-) mouse modelDisrupted synaptic development in the hypoxic newborn brain.Calpain inhibition reduces amplitude and accelerates decay of the late sodium current in ventricular myocytes from dogs with chronic heart failurePost-transcriptional silencing of SCN1B and SCN2B genes modulates late sodium current in cardiac myocytes from normal dogs and dogs with chronic heart failure.LQT4 gene: the "missing" ankyrin.Defects in cytoskeletal signaling pathways, arrhythmia, and sudden cardiac death.The human heart and rat brain IIA Na+ channels interact with different molecular regions of the beta1 subunitLate sodium current in failing heart: friend or foe?Late sodium current is a new therapeutic target to improve contractility and rhythm in failing heart.Late Na+ current produced by human cardiac Na+ channel isoform Nav1.5 is modulated by its beta1 subunit.Molecular mechanisms of inherited arrhythmias.Electrophysiology and beyond: multiple roles of Na+ channel β subunits in development and disease.The spectrin-ankyrin-4.1-adducin membrane skeleton: adapting eukaryotic cells to the demands of animal life.Voltage-gated sodium channels: biophysics, pharmacology, and related channelopathies.An introduction to murine models of atrial fibrillationAutoimmune antigenic targets at the node of Ranvier in demyelinating disorders.Molecular Pathophysiology of Congenital Long QT Syndrome.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.Cytoskeletal basis of ion channel function in cardiac muscle.Long QT syndrome: from channels to cardiac arrhythmias.Cytoskeletal actin microfilaments and the transient outward potassium current in hypertrophied rat ventriculocytes.Distinct subcellular mechanisms for the enhancement of the surface membrane expression of SK2 channel by its interacting proteins, α-actinin2 and filamin A.Identification of Ank(G107), a muscle-specific ankyrin-G isoform.L-type but not T-type calcium current changes during postnatal development in rabbit sinoatrial node.The Effect of Astragaloside on Pacemaker Current and the Cytoskeleton in Rabbit Sinoatrial Node Cells Under the Ischemia and Reperfusion Condition.Lost anchors cost lives
P2860
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P2860
Abnormal cardiac Na(+) channel properties and QT heart rate adaptation in neonatal ankyrin(B) knockout mice.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
2000年學術文章
@zh-hant
name
Abnormal cardiac Na
@nl
Abnormal cardiac Na(+) channel ...... atal ankyrin(B) knockout mice.
@en
type
label
Abnormal cardiac Na
@nl
Abnormal cardiac Na(+) channel ...... atal ankyrin(B) knockout mice.
@en
prefLabel
Abnormal cardiac Na
@nl
Abnormal cardiac Na(+) channel ...... atal ankyrin(B) knockout mice.
@en
P2093
P356
P1433
P1476
Abnormal cardiac Na(+) channel ...... atal ankyrin(B) knockout mice.
@en
P2093
P304
P356
10.1161/01.RES.86.4.441
P577
2000-03-01T00:00:00Z