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Regulation of the Pax6 : Pax6(5a) mRNA ratio in the developing mammalian brain.Foxg1 is required for specification of ventral telencephalon and region-specific regulation of dorsal telencephalic precursor proliferation and apoptosisLoss of Wnt8b has no overt effect on hippocampus development but leads to altered Wnt gene expression levels in dorsomedial telencephalonThe winged helix transcription factor Foxg1 facilitates retinal ganglion cell axon crossing of the ventral midline in the mousePax6 exerts regional control of cortical progenitor proliferation via direct repression of Cdk6 and hypophosphorylation of pRbAnalysis of early ventral telencephalic defects in mice lacking functional Gli3 proteinComparison of two related lines of tauGFP transgenic mice designed for lineage tracingFunctional conservation of Pax6 regulatory elements in humans and mice demonstrated with a novel transgenic reporter mousePositive autoregulation of the transcription factor Pax6 in response to increased levels of either of its major isoforms, Pax6 or Pax6(5a), in cultured cellsNewly identified patterns of Pax2 expression in the developing mouse forebrainNovel lines of Pax6-/- embryonic stem cells exhibit reduced neurogenic capacity without loss of viabilityWnt/β-catenin signalling is active in a highly dynamic pattern during development of the mouse cerebellum.The expression and activity of β-catenin in the thalamus and its projections to the cerebral cortex in the mouse embryo.Activation of Wnt/β-catenin signalling affects differentiation of cells arising from the cerebellar ventricular zoneScreening for mammalian neural genes via fluorescence-activated cell sorter purification of neural precursors from Sox1-gfp knock-in mice.The transcription factor Foxg1 regulates telencephalic progenitor proliferation cell autonomously, in part by controlling Pax6 expression levels.Identification of genomic regions regulating Pax6 expression in embryonic forebrain using YAC reporter transgenic mouse lines.Cerebral Cortex Expression of Gli3 Is Required for Normal Development of the Lateral Olfactory TractExpression of Barhl2 and its relationship with Pax6 expression in the forebrain of the mouse embryo.Lateral cortical Cdca7 expression levels are regulated by Pax6 and influence the production of intermediate progenitorsControlled overexpression of Pax6 in vivo negatively autoregulates the Pax6 locus, causing cell-autonomous defects of late cortical progenitor proliferation with little effect on cortical arealizationAbnormal positioning of diencephalic cell types in neocortical tissue in the dorsal telencephalon of mice lacking functional Gli3.Long-range downstream enhancers are essential for Pax6 expression.Normal ventral telencephalic expression of Pax6 is required for normal development of thalamocortical axons in embryonic mice.Analysis of compound heterozygotes reveals that the mouse floxed Pax6 (tm1Ued) allele produces abnormal eye phenotypes.Heparan sulfotransferases Hs6st1 and Hs2st keep Erk in check for mouse corpus callosum development.Regulation of cerebral cortical neurogenesis by the Pax6 transcription factor.Analysis of axon guidance defects at the optic chiasm in heparan sulphate sulphotransferase compound mutant mice.The transcription factor Foxg1 regulates the competence of telencephalic cells to adopt subpallial fates in mice.Carcinogen-induced pancreatic lesions in the mouse: effect of Smad4 and Apc genotypes.Hybridization parameters revisited: solutions containing SDS.Specific modification of heparan sulphate is required for normal cerebral cortical development.Disruption of early events in thalamocortical tract formation in mice lacking the transcription factors Pax6 or Foxg1.Heparan sulphation patterns generated by specific heparan sulfotransferase enzymes direct distinct aspects of retinal axon guidance at the optic chiasm.Expansion of the piriform cortex contributes to corticothalamic pathfinding defects in Gli3 conditional mutants.Evaluation of the mouse TgTP6.3 tauGFP transgene as a lineage marker in chimeras.Pax3 regulates Wnt1 expression via a conserved binding site in the 5' proximal promoter.Heparan sulfate sugar modifications mediate the functions of slits and other factors needed for mouse forebrain commissure development.Embryonic stem cells and transgenic mice ubiquitously expressing a tau-tagged green fluorescent protein.Wnt/β-catenin signaling is disrupted in the extra-toes (Gli3(Xt/Xt) ) mutant from early stages of forebrain development, concomitant with anterior neural plate patterning defects.
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
John O Mason
@ast
John O Mason
@en
John O Mason
@es
John O Mason
@nl
John O Mason
@sl
type
label
John O Mason
@ast
John O Mason
@en
John O Mason
@es
John O Mason
@nl
John O Mason
@sl
prefLabel
John O Mason
@ast
John O Mason
@en
John O Mason
@es
John O Mason
@nl
John O Mason
@sl
P1053
A-1926-2010
P106
P31
P3829
P496
0000-0002-0489-2400