Corticosterone selectively targets endo-cortical surfaces by an osteoblast-dependent mechanism.
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Glucocorticoid receptor signaling in health and diseaseQuiescent Bone Lining Cells Are a Major Source of Osteoblasts During Adulthood.Disruption of glucocorticoid signaling in chondrocytes delays metaphyseal fracture healing but does not affect normal cartilage and bone developmentEvaluation of the effects of LLLT on biomechanical properties of tibial diaphysis in two rat models of experimental osteoporosis by a three point bending test.Evaluating glucocorticoid administration on biomechanical properties of rats' tibial diaphysisEndogenous Glucocorticoids and Bone.Supraphysiologic glucocorticoid administration increased biomechanical bone strength of rats' vertebral body.Osteoblasts mediate the adverse effects of glucocorticoids on fuel metabolism.The multiple facets of glucocorticoid action in rheumatoid arthritis.Osteocalcin: an osteoblast-derived polypeptide hormone that modulates whole body energy metabolism.Blockade of interleukin-6 receptor enhances the anti-arthritic effect of glucocorticoids without decreasing bone mineral density in mice with collagen-induced arthritis.The biochemical signatures of stress: A preliminary analysis of osteocalcin concentrations and macroscopic skeletal changes associated with stress in the 13th - 17th centuries black friars population.New mechanisms of glucocorticoid-induced insulin resistance: make no bones about it.Cortical bone loss caused by glucocorticoid excess requires RANKL production by osteocytes and is associated with reduced OPG expression in mice.Acute effects of glucocorticoids on serum markers of osteoclasts, osteoblasts, and osteocytes.TET2 Loss Dysregulates the Behavior of Bone Marrow Mesenchymal Stromal Cells and Accelerates Tet2-/--Driven Myeloid Malignancy Progression.Evaluation of the anti-osteoporotic effect of Ginkgo biloba L. in Wistar rats with glucocorticoid-induced-osteoporosis by bone densitometry using dual-energy x-ray absorptiometry (DEXA) and mechanical testing.SLE disease per se contributes to deterioration in bone mineral density, microstructure and bone strength.The effect of the Ginkgo biloba extract in the expression of Bax, Bcl-2 and bone mineral content of Wistar rats with glucocorticoid-induced osteoporosis.
P2860
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P2860
Corticosterone selectively targets endo-cortical surfaces by an osteoblast-dependent mechanism.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
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2011年學術文章
@zh-hant
name
Corticosterone selectively tar ...... steoblast-dependent mechanism.
@en
Corticosterone selectively tar ...... steoblast-dependent mechanism.
@nl
type
label
Corticosterone selectively tar ...... steoblast-dependent mechanism.
@en
Corticosterone selectively tar ...... steoblast-dependent mechanism.
@nl
prefLabel
Corticosterone selectively tar ...... steoblast-dependent mechanism.
@en
Corticosterone selectively tar ...... steoblast-dependent mechanism.
@nl
P2093
P50
P1433
P1476
Corticosterone selectively tar ...... steoblast-dependent mechanism.
@en
P2093
Dörte Huscher
Frank Buttgereit
Markus Herrmann
Nicky Bertollo
Robert Kalak
Tara C Brennan-Speranza
Uta Heinevetter
P304
P356
10.1016/J.BONE.2011.06.013
P577
2011-06-22T00:00:00Z