about
Disruption of glucocorticoid signaling in chondrocytes delays metaphyseal fracture healing but does not affect normal cartilage and bone developmentOsteoblasts mediate the adverse effects of glucocorticoids on fuel metabolism.Glucocorticoids and bone: local effects and systemic implications.Continuous corticosterone delivery via the drinking water or pellet implantation: A comparative study in mice.Long-term corticosterone treatment induced lobe-specific pathology in mouse prostate.The challenge of continuous exogenous glucocorticoid administration in mice.Association of adipokines and joint biomarkers with cartilage-modifying effects of weight loss in obese subjects.Chronic Mild Stress Causes Bone Loss via an Osteoblast-Specific Glucocorticoid-Dependent Mechanism.Corticosterone selectively targets endo-cortical surfaces by an osteoblast-dependent mechanism.Sclerostin Blockade-A Dual Mode of Action After All?Ubiquitous expression of the Pik3caH1047R mutation promotes hypoglycemia, hypoinsulinemia, and organomegaly.β-Blockers and bone healthLeopard skinAndrogens sensitise mice to glucocorticoid-induced insulin resistance and fat accumulationDisruption of glucocorticoid signalling in osteoblasts attenuates age-related surgically induced osteoarthritis
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Holger Henneicke
@ast
Holger Henneicke
@en
Holger Henneicke
@es
Holger Henneicke
@nl
Holger Henneicke
@sl
type
label
Holger Henneicke
@ast
Holger Henneicke
@en
Holger Henneicke
@es
Holger Henneicke
@nl
Holger Henneicke
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prefLabel
Holger Henneicke
@ast
Holger Henneicke
@en
Holger Henneicke
@es
Holger Henneicke
@nl
Holger Henneicke
@sl
P106
P31
P496
0000-0003-1746-9369