TRPC6 and TRPC4 Heteromultimerization Mediates Store Depletion-Activated NCX1 Reversal in Proliferative Vascular Smooth Muscle Cells.

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TRPC6 and TRPC4 Heteromultimerization Mediates Store Depletion-Activated NCX1 Reversal in Proliferative Vascular Smooth Muscle Cells.

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http://www.wikidata.org/entity/Q52725298

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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name

TRPC6 and TRPC4 Heteromultimer ...... Vascular Smooth Muscle Cells.

@en

TRPC6 and TRPC4 Heteromultimer ...... Vascular Smooth Muscle Cells.

@nl

type

Item

label

TRPC6 and TRPC4 Heteromultimer ...... Vascular Smooth Muscle Cells.

@en

TRPC6 and TRPC4 Heteromultimer ...... Vascular Smooth Muscle Cells.

@nl

prefLabel

TRPC6 and TRPC4 Heteromultimer ...... Vascular Smooth Muscle Cells.

@en

TRPC6 and TRPC4 Heteromultimer ...... Vascular Smooth Muscle Cells.

@nl

P1476

TRPC6 and TRPC4 Heteromultimer ...... Vascular Smooth Muscle Cells.

@en

P2093

Bei Liu

http://www.w3.org/2001/XMLSchema#string

Bin Zhang

http://www.w3.org/2001/XMLSchema#string

Carolyn M Roos

http://www.w3.org/2001/XMLSchema#string

Jordan D Miller

http://www.w3.org/2001/XMLSchema#string

Michael A Thompson

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Rui-Wei Guo

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Y S Prakash

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P2860

STIM1 heteromultimerizes TRPC channels to determine their function as store-operated channels

Junctophilin-4, a component of the endoplasmic reticulum-plasma membrane junctions, regulates Ca2+ dynamics in T cells

Reactive oxygen species-mediated TRPC6 protein activation in vascular myocytes, a mechanism for vasoconstrictor-regulated vascular tone.

Sodium-calcium exchange in intracellular calcium handling of human airway smooth muscle

STIM1-dependent and STIM1-independent function of transient receptor potential canonical (TRPC) channels tunes their store-operated mode.

Leukotriene-C4 synthase, a critical enzyme in the activation of store-independent Orai1/Orai3 channels, is required for neointimal hyperplasia.

Evidence for STIM1- and Orai1-dependent store-operated calcium influx through ICRAC in vascular smooth muscle cells: role in proliferation and migration.

Molecular determinants mediating gating of Transient Receptor Potential Canonical (TRPC) channels by stromal interaction molecule 1 (STIM1).

Nanomolar potency and selectivity of a Ca²⁺ release-activated Ca²⁺ channel inhibitor against store-operated Ca²⁺ entry and migration of vascular smooth muscle cells.

Nonsteroidal anti-inflammatory drugs inhibit vascular smooth muscle cell proliferation by enabling the Ca2+-dependent inactivation of calcium release-activated calcium/orai channels normally prevented by mitochondria.

P304

119-125

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scholarly article

P356

10.1080/19336950.2018.1451696

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P478

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2018-01-01T00:00:00Z

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29560783

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5972809

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