Vitamin E decreases bone mass by stimulating osteoclast fusion. - Wikidata - wikimedia - TriplyDB

Vitamin E decreases bone mass by stimulating osteoclast fusion.

Vitamin E decreases bone mass by stimulating osteoclast fusion.

http://www.wikidata.org/entity/Q53172611

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Activin A inhibits RANKL-mediated osteoclast formation, movement and function in murine bone marrow macrophage cultures.Retinoid X receptors orchestrate osteoclast differentiation and postnatal bone remodeling.The effects of α-tocopherol on bone: a double-edged sword?Tissue distribution of emulsified γ-tocotrienol and its long-term biological effects after subcutaneous administration.Effects of annatto-derived tocotrienol supplementation on osteoporosis induced by testosterone deficiency in rats.Annatto tocotrienol improves indices of bone static histomorphometry in osteoporosis due to testosterone deficiency in rats.Comparative gene-expression analysis of the dental follicle and periodontal ligament in humans.Clinical Significance of Female-hormones and Cytokines in Breast Cancer Patients Complicated with Aromatase Inhibitor-related Osteoarthropathy - Efficacy of Vitamin E.The biological effects of tocotrienol on bone: a review on evidence from rodent models.Deletion of histone deacetylase 7 in osteoclasts decreases bone mass in mice by interactions with MITF.Sexual Dimorphism in MAPK-Activated Protein Kinase-2 (MK2) Regulation of RANKL-Induced Osteoclastogenesis in Osteoclast Progenitor Subpopulations.High-Dose α-Tocopherol Supplementation Does Not Induce Bone Loss in Normal Rats.Vitamin E as an Antiosteoporotic Agent via Receptor Activator of Nuclear Factor Kappa-B Ligand Signaling Disruption: Current Evidence and Other Potential Research Areas The genetic pleiotropy of musculoskeletal aging.Selenium- and Tellurium-Based Antioxidants for Modulating Inflammation and Effects on Osteoblastic Activity Palm tocotrienol supplementation enhanced bone formation in oestrogen-deficient rats.Vitamin e and the healing of bone fracture: the current state of evidence Osteoclast fusion and regulation by RANKL-dependent and independent factors.Advances in osteoclast biology reveal potential new drug targets and new roles for osteoclasts Vitamin E slows down the progression of osteoarthritis.Advances in the regulation of osteoclasts and osteoclast functions.Effects of vitamin e on bone biomechanical and histomorphometric parameters in ovariectomized rats α-Tocopheryl Succinate Inhibits Osteoclast Formation by Suppressing Receptor Activator of Nuclear Factor-kappaB Ligand (RANKL) Expression and Bone Resorption.Vitamin D receptor expression in human bone tissue and dose-dependent activation in resorbing osteoclasts.Zinc inhibits osteoclast differentiation by suppression of Ca2+-Calcineurin-NFATc1 signaling pathway Evaluation of long-term vitamin E insufficiency or excess on bone mass, density, and microarchitecture in rodents.Intake and serum concentrations of α-tocopherol in relation to fractures in elderly women and men: 2 cohort studies.Interactions between α-tocopherol, polyunsaturated fatty acids, and lipoxygenases during embryogenesis.The Effects of Annatto Tocotrienol on Bone Biomechanical Strength and Bone Calcium Content in an Animal Model of Osteoporosis Due to Testosterone Deficiency.p38α MAPK regulates proliferation and differentiation of osteoclast progenitors and bone remodeling in an aging-dependent manner.Bioavailability of vitamin E in humans: an update.STATs and macrophage fusion.Associations between serum vitamin E concentration and bone mineral density in the US elderly population.MicroRNA-145 regulates osteoblastic differentiation by targeting the transcription factor Cbfb.Pin1, the Master Orchestrator of Bone Cell Differentiation.Bisphosphonates inhibit osteosarcoma-mediated osteolysis via attenuation of tumor expression of MCP-1 and RANKL.Partial Protection by Dietary Antioxidants Against Ethanol-Induced Osteopenia and Changes in Bone Morphology in Female Mice.The multi-faceted role of retinoid X receptor in bone remodeling.Circadian Clock Regulates Bone Resorption in Mice.Alpha-tocopherol attenuates the anti-tumor activity of crizotinib against cells transformed by NPM-ALK

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Vitamin E decreases bone mass by stimulating osteoclast fusion.

http://www.wikidata.org/entity/Q53172611

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2012 nî lūn-bûn

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name

Vitamin E decreases bone mass by stimulating osteoclast fusion.

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Vitamin E decreases bone mass by stimulating osteoclast fusion.

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Vitamin E decreases bone mass by stimulating osteoclast fusion.

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Vitamin E decreases bone mass by stimulating osteoclast fusion.

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prefLabel

Vitamin E decreases bone mass by stimulating osteoclast fusion.

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Vitamin E decreases bone mass by stimulating osteoclast fusion.

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Vitamin E decreases bone mass by stimulating osteoclast fusion.

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Atsushi Okawa

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Chengshan Ma

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Hiroki Ochi

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Hiroshi Itoh

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Hiroshi Takayanagi

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Hiroshi Tamai

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Hiroyuki Arai

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Kenichi Shinomiya

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Kimitaka Takitani

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Koji Fujita

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Mitf and Tfe3, two members of the Mitf-Tfe family of bHLH-Zip transcription factors, have important but functionally redundant roles in osteoclast development.

The p38 MAPK pathway is essential for skeletogenesis and bone homeostasis in mice

Osteoimmunology: interactions of the bone and immune system

Up-regulation of c-Jun-NH2-kinase pathway contributes to the induction of mitochondria-mediated apoptosis by alpha-tocopheryl succinate in human prostate cancer cells.

Meta-analysis: high-dosage vitamin E supplementation may increase all-cause mortality

Leptin inhibits bone formation through a hypothalamic relay: a central control of bone mass

DC-STAMP is essential for cell-cell fusion in osteoclasts and foreign body giant cells

Alpha-tocopherol transfer protein is important for the normal development of placental labyrinthine trophoblasts in mice

Osteoclast stimulatory transmembrane protein (OC-STAMP), a novel protein induced by RANKL that promotes osteoclast differentiation

Molecules and signaling pathways involved in the expression of OC-STAMP during osteoclastogenesis

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589-594

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10.1038/NM.2659

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4

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22388090

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