Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

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Allyl Isothiocyanate Exhibits No Anticancer Activity in MDA-MB-231 Breast Cancer Cells.

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Q47233186-2E9DACA2-EE78-45A0-B716-D81DE887F479

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Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

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http://www.wikidata.org/entity/Q53223354

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2015 nî lūn-bûn

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Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

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Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

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label

Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

@en

Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

@nl

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Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

@en

Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

@nl

P1476

Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

@en

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Armanda Pugnaloni

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Corrado Rubini

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Elisabetta Strafella

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Ernesto Pasquini

http://www.w3.org/2001/XMLSchema#string

Linda Nocchi

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Marco Tomasetti

http://www.w3.org/2001/XMLSchema#string

Massimo Re

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Matteo Valentino

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Monica Amati

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Nicola Manzella

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P2860

EGFR nuclear translocation modulates DNA repair following cisplatin and ionizing radiation treatment

Accumulation of premutagenic DNA lesions in mice defective in removal of oxidative base damage

The human OGG1 gene: structure, functions, and its implication in the process of carcinogenesis

Identification of the tuberous sclerosis complex-2 tumor suppressor gene product tuberin as a target of the phosphoinositide 3-kinase/akt pathway

Molecular mechanism of regulation of OGG1: tuberin deficiency results in cytoplasmic redistribution of transcriptional factor NF-YA.

Hyperactivation of Akt/mTOR and deficiency in tuberin increased the oxidative DNA damage in kidney cancer patients with diabetes.

Carcinogenic effects of wood dust: review and discussion.

RETRACTED: Nrf2/p62 signaling in apoptosis resistance and its role in cadmium-induced carcinogenesis

Regulation of autophagy by mTOR-dependent and mTOR-independent pathways: autophagy dysfunction in neurodegenerative diseases and therapeutic application of autophagy enhancers.

Novel mechanism of regulation of the DNA repair enzyme OGG1 in tuberin-deficient cells.

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487-497

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scholarly article

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10.1093/MUTAGE/GEV007

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P478

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Massimo Bracci

Lory Santarelli

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2015-02-23T00:00:00Z

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P5875

272841738

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25711499

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Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

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Wood dust exposure induces cell transformation through EGFR-mediated OGG1 inhibition.

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P1476

P2093

P2860

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P577

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P698