Level of activation of the unfolded protein response correlates with Paneth cell apoptosis in human small intestine exposed to ischemia/reperfusion.
about
Paneth cell α-defensins and enteric microbiota in health and diseaseLife and death at the mucosal-luminal interface: New perspectives on human intestinal ischemia-reperfusionRole of endoplasmic reticulum stress and autophagy as interlinking pathways in the pathogenesis of inflammatory bowel diseaseGoblet cell compound exocytosis in the defense against bacterial invasion in the colon exposed to ischemia-reperfusionIntestinal permeability--a new target for disease prevention and therapyIntegration of UPRER and oxidative stress signaling in the control of intestinal stem cell proliferationPaneth cell extrusion and release of antimicrobial products is directly controlled by immune cell-derived IFN-γ.Real-time in vivo imaging of early mucosal changes during ischemia-reperfusion in human jejunumPretreatment with the ALDH2 agonist Alda-1 reduces intestinal injury induced by ischaemia and reperfusion in mice.The cell biology of the unfolded protein response.Starvation compromises Paneth cells.Paneth cell ablation in the presence of Klebsiella pneumoniae induces necrotizing enterocolitis (NEC)-like injury in the small intestine of immature mice.The unfolded protein response and its role in intestinal homeostasis and inflammation.Autophagy, microbial sensing, endoplasmic reticulum stress, and epithelial function in inflammatory bowel diseasePaneth cell-mediated multiorgan dysfunction after acute kidney injury.The unfolded protein response and gastrointestinal diseaseEffect of liposome-encapsulated hemoglobin resuscitation on proteostasis in small intestinal epithelium after hemorrhagic shock.Effects of n-3 PUFAs on Intestinal Mucosa Innate Immunity and Intestinal Microbiota in Mice after Hemorrhagic Shock ResuscitationDeficiency in Duox2 activity alleviates ileitis in GPx1- and GPx2-knockout mice without affecting apoptosis incidence in the crypt epithelium.High Fat Diet Alters Gut Microbiota and the Expression of Paneth Cell-Antimicrobial Peptides Preceding Changes of Circulating Inflammatory Cytokines.The role of innate immune-stimulated epithelial apoptosis during gastrointestinal inflammatory diseases.Oxygen in the regulation of intestinal epithelial transport.Animal models of ischemia-reperfusion-induced intestinal injury: progress and promise for translational research.Critical role of interleukin-17A in murine intestinal ischemia-reperfusion injury.Berberine ameliorates pro-inflammatory cytokine-induced endoplasmic reticulum stress in human intestinal epithelial cells in vitro.Dysbiosis of intestinal microbiota and decrease in paneth cell antimicrobial peptide level during acute necrotizing pancreatitis in rats.Rapid lamina propria retraction and zipper-like constriction of the epithelium preserves the epithelial lining in human small intestine exposed to ischaemia-reperfusion."ER stress(ed out)!": Paneth cells and ischemia-reperfusion injury of the small intestine.Paneth Cell Alterations in the Development and Phenotype of Crohn's Disease.Paneth cells in intestinal physiology and pathophysiology.Paneth and intestinal stem cells preserve their functional integrity during worsening of acute cellular rejection in small bowel transplantation.Nurr1 promotes intestinal regeneration after ischemia/reperfusion injury by inhibiting the expression of p21 (Waf1/Cip1).Innate immune responses to trauma.Endoplasmic reticulum stress of Kupffer cells involved in the conversion of natural regulatory T cells to Th17 cells in liver ischemia-reperfusion injury.Reduced Paneth cell antimicrobial protein levels correlate with activation of the unfolded protein response in the gut of obese individuals.Targeting the miR-665-3p-ATG4B-autophagy axis relieves inflammation and apoptosis in intestinal ischemia/reperfusion.Innate immunity in the small intestineComplement After Trauma: Suturing Innate and Adaptive Immunity
P2860
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P2860
Level of activation of the unfolded protein response correlates with Paneth cell apoptosis in human small intestine exposed to ischemia/reperfusion.
description
2010 nî lūn-bûn
@nan
2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
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2010年學術文章
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name
Level of activation of the unf ...... posed to ischemia/reperfusion.
@en
Level of activation of the unf ...... posed to ischemia/reperfusion.
@nl
type
label
Level of activation of the unf ...... posed to ischemia/reperfusion.
@en
Level of activation of the unf ...... posed to ischemia/reperfusion.
@nl
prefLabel
Level of activation of the unf ...... posed to ischemia/reperfusion.
@en
Level of activation of the unf ...... posed to ischemia/reperfusion.
@nl
P2093
P1433
P1476
Level of activation of the unf ...... posed to ischemia/reperfusion.
@en
P2093
Caroline M Hodin
Cornelis H C Dejong
Fons K Verheyen
Jacco-Juri de Haan
Joep Grootjans
Joep P M Derikx
Ronald M van Dam
Wim A Buurman
P304
529-539.e3
P356
10.1053/J.GASTRO.2010.10.040
P407
P577
2010-10-19T00:00:00Z