N-ras oncogene causes AP-2 transcriptional self-interference, which leads to transformation.
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A family of AP-2 proteins down-regulate manganese superoxide dismutase expressionTranscription factor AP-2 regulates human insulin-like growth factor binding protein-5 gene expressionRB and c-Myc activate expression of the E-cadherin gene in epithelial cells through interaction with transcription factor AP-2.Transcription factor AP-2alpha is preferentially cleaved by caspase 6 and degraded by proteasome during tumor necrosis factor alpha-induced apoptosis in breast cancer cellsTranscriptional regulation of the AP-2alpha promoter by BTEB-1 and AP-2rep, a novel wt-1/egr-related zinc finger repressorTranscriptional activation by Myc is under negative control by the transcription factor AP-2Activator protein 2alpha suppresses intestinal tumorigenesis in the Apc(min) mouseActivator protein 2alpha associates with adenomatous polyposis coli/beta-catenin and Inhibits beta-catenin/T-cell factor transcriptional activity in colorectal cancer cellsCloning and characterization of a novel mouse AP-2 transcription factor, AP-2delta, with unique DNA binding and transactivation propertiesEnhanced apoptotic cell death of renal epithelial cells in mice lacking transcription factor AP-2betaFunctional interaction between a RARE and an AP-2 binding site in the regulation of the human HOX A4 gene promoterThe Kruppel-like KLF4 transcription factor, a novel regulator of urokinase receptor expression, drives synthesis of this binding site in colonic crypt luminal surface epithelial cells.Ku proteins interact with activator protein-2 transcription factors and contribute to ERBB2 overexpression in breast cancer cell lines.Loss of AP-2 results in downregulation of c-KIT and enhancement of melanoma tumorigenicity and metastasis.Induction of AP-2alpha expression by adenoviral infection involves inactivation of the AP-2rep transcriptional corepressor CtBP1.Coactivator PC4 mediates AP-2 transcriptional activity and suppresses ras-induced transformation dependent on AP-2 transcriptional interference.Novel promoter sequence required for manganese regulation of manganese peroxidase isozyme 1 gene expression in Phanerochaete chrysosporium.Yin Yang 1 cooperates with activator protein 2 to stimulate ERBB2 gene expression in mammary cancer cells.Comparative and functional analysis of the AP2 promoter indicates that conserved octamer and initiator elements are critical for activity.The developmentally regulated transcription factor AP-2 is involved in c-erbB-2 overexpression in human mammary carcinoma.Reversal of epigenetic silencing of AP-2alpha results in increased zinc uptake in DU-145 and LNCaP prostate cancer cells.Activation of the mitogen-activated protein kinase pathway induces transcription of the PAC-1 phosphatase gene.Expression of transcription factor AP-2alpha predicts survival in epithelial ovarian cancerExtracellular matrix fibronectin increases prostaglandin E2 receptor subtype EP4 in lung carcinoma cells through multiple signaling pathways: the role of AP-2.Transactivation of the urokinase-type plasminogen activator receptor gene through a novel promoter motif bound with an activator protein-2alpha-related factor.AP2 suppresses osteoblast differentiation and mineralization through down-regulation of Frizzled-1.Hepatitis B virus regulation of Raf1 promoter activity through activation of transcription factor AP-2α.The retinoblastoma protein binds the promoter of the survival gene bcl-2 and regulates its transcription in epithelial cells through transcription factor AP-2.PolyADP-ribose polymerase is a coactivator for AP-2-mediated transcriptional activation.Apoptosis induction by activator protein 2alpha involves transcriptional repression of Bcl-2.Dual regulation of AP-2alpha transcriptional activation by poly(ADP-ribose) polymerase-1The hepatitis B virus X protein inhibits secretion of apolipoprotein B by enhancing the expression of N-acetylglucosaminyltransferase III.Cell cycle arrest and apoptosis induction by activator protein 2alpha (AP-2alpha) and the role of p53 and p21WAF1/CIP1 in AP-2alpha-mediated growth inhibition.Tumorigenic effect of transcription factor hAP-2alpha and the intricate link between hAP-2alpha activation and squelching.Dominant negative interference of transcription factor AP-2 causes inhibition of ErbB-3 expression and suppresses malignant cell growth.Transcription factor AP-2 functions as a repressor that contributes to the liver-specific expression of serum amyloid A1 gene.Dual role for transcription factor AP-2 in the regulation of the major fetal promoter P3 of the gene for human insulin-like growth factor II.Identification and characterization of the cis-acting elements of the human CD155 gene core promoter.Heregulin-dependent autocrine loop regulates growth of K-ras but not erbB-2 transformed rat thyroid epithelial cells.Regulation of the mouse cartilage-derived retinoic acid-sensitive protein gene by the transcription factor AP-2.
P2860
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P2860
N-ras oncogene causes AP-2 transcriptional self-interference, which leads to transformation.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
1994年學術文章
@zh
1994年學術文章
@zh-hant
name
N-ras oncogene causes AP-2 tra ...... which leads to transformation.
@en
N-ras oncogene causes AP-2 tra ...... which leads to transformation.
@nl
type
label
N-ras oncogene causes AP-2 tra ...... which leads to transformation.
@en
N-ras oncogene causes AP-2 tra ...... which leads to transformation.
@nl
prefLabel
N-ras oncogene causes AP-2 tra ...... which leads to transformation.
@en
N-ras oncogene causes AP-2 tra ...... which leads to transformation.
@nl
P2093
P356
P1433
P1476
N-ras oncogene causes AP-2 tra ...... which leads to transformation
@en
P2093
P304
P356
10.1101/GAD.8.11.1258
P50
P577
1994-06-01T00:00:00Z