Fructose, but not dextrose, accelerates the progression of chronic kidney disease. - Wikidata - wikimedia - TriplyDB

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

http://www.wikidata.org/entity/Q53536824

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Opposing effects of fructokinase C and A isoforms on fructose-induced metabolic syndrome in mice Animal models for the study of arterial hypertension Direct renal effects of a fructose-enriched diet: interaction with high salt intake Long term metabolic syndrome induced by a high fat high fructose diet leads to minimal renal injury in C57BL/6 mice Both isoforms of ketohexokinase are dispensable for normal growth and development Fructose Intake, Serum Uric Acid, and Cardiometabolic Disorders: A Critical Review.The Role of Overweight and Obesity in the Cardiorenal Syndrome.Role of the immune system in hypertension: modulation by dietary antioxidants.Sugary soda consumption and albuminuria: results from the National Health and Nutrition Examination Survey, 1999-2004.Moderate (20%) fructose-enriched diet stimulates salt-sensitive hypertension with increased salt retention and decreased renal nitric oxide Comparison of free fructose and glucose to sucrose in the ability to cause fatty liver High Dietary Fructose: Direct or Indirect Dangerous Factors Disturbing Tissue and Organ Functions Dietary fructose inhibits intestinal calcium absorption and induces vitamin D insufficiency in CKD.Dietary fructose causes tubulointerstitial injury in the normal rat kidney The Effect of Diet on the Survival of Patients with Chronic Kidney Disease.Increased fructose consumption is associated with fibrosis severity in patients with nonalcoholic fatty liver disease Effect of chenodeoxycholic acid on fibrosis, inflammation and oxidative stress in kidney in high-fructose-fed Wistar rats.Endogenous fructose production and fructokinase activation mediate renal injury in diabetic nephropathy.Sugary drinks in the pathogenesis of obesity and cardiovascular diseases.Hypothesis: could excessive fructose intake and uric acid cause type 2 diabetes?The Relation between Fructose-Induced Metabolic Syndrome and Altered Renal Haemodynamic and Excretory Function in the Rat.The impact of fructose on renal function and blood pressure.Renoprotective and antioxidant effects of Saururus chinensis Baill in rats fed a high-fructose diet.Sugar-sweetened soda consumption, hyperuricemia, and kidney disease.A Long-Term High-Fat/High-Sucrose Diet Promotes Kidney Lipid Deposition and Causes Apoptosis and Glomerular Hypertrophy in Bama Minipigs.The Envy of Scholars: Applying the Lessons of the Framingham Heart Study to the Prevention of Chronic Kidney Disease Low-fructose diet lowers blood pressure and inflammation in patients with chronic kidney disease.Bioactivity-Guided Identification of Botanical Inhibitors of Ketohexokinase Health implications of high-fructose intake and current research.Fructokinase, Fructans, Intestinal Permeability, and Metabolic Syndrome: An Equine Connection?Synergistic effect of uricase blockade plus physiological amounts of fructose-glucose on glomerular hypertension and oxidative stress in rats.Fructose and uric acid in diabetic nephropathy In vivo Antihypertensive and Antihyperlipidemic Effects of the Crude Extracts and Fractions of Moringa stenopetala (Baker f.) Cufod. Leaves in Rats Fructose induces the inflammatory molecule ICAM-1 in endothelial cells.Excessive fructose intake causes 1,25-(OH)(2)D(3)-dependent inhibition of intestinal and renal calcium transport in growing rats.Urine NGAL and KIM-1 in children and adolescents with hyperuricemia Cinnamaldehyde Attenuates Cataractogenesis via Restoration of Hypertension and Oxidative Stress in Fructose-Fed Hypertensive rats Potential role of uric acid in metabolic syndrome, hypertension, kidney injury, and cardiovascular diseases: is it time for reappraisal?Ketohexokinase-dependent metabolism of fructose induces proinflammatory mediators in proximal tubular cells.Endogenous fructose production and metabolism in the liver contributes to the development of metabolic syndrome.

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P2860

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

http://www.wikidata.org/entity/Q53536824

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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name

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

@en

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

@nl

type

label

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

@en

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

@nl

prefLabel

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

@en

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

@nl

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AJPRENAL.00181.2007

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American Journal of Physiology - Renal Physiology

P1476

Fructose, but not dextrose, accelerates the progression of chronic kidney disease.

@en

P2093

Carlos Roncal

http://www.w3.org/2001/XMLSchema#string

Li Zhang

http://www.w3.org/2001/XMLSchema#string

Michael S Gersch

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Pietro Cirillo

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Richard J Johnson

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Sirirat Reungjui

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Takahiko Nakagawa

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Wei Mu

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Yuri Y Sautin

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P2860

D-tagatose, a stereoisomer of D-fructose, increases blood uric acid concentration

Nutritional significance of fructose and sugar alcohols.

Amelioration of insulin resistance and hypertension in a fructose-fed rat model with fish oil supplementation.

A causal role for uric acid in fructose-induced metabolic syndrome.

Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-beta antibody in db/db diabetic mice.

Prevention of fructose-induced hypertension by dietary vitamins.

IL-1 up-regulates osteopontin expression in experimental crescentic glomerulonephritis in the rat.

Discovery of protein biomarkers for renal diseases.

Insulin resistance, hyperinsulinemia, and renal injury: mechanisms and implications.

Hypothesis: fructose-induced hyperuricemia as a causal mechanism for the epidemic of the metabolic syndrome.

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6168231

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17670904

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