The effect of peripheral nerve injury on disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
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An ENU-induced mutation in mouse glycyl-tRNA synthetase (GARS) causes peripheral sensory and motor phenotypes creating a model of Charcot-Marie-Tooth type 2D peripheral neuropathyMolecular chaperone mediated late-stage neuroprotection in the SOD1(G93A) mouse model of amyotrophic lateral sclerosisCo-induction of the heat shock response ameliorates disease progression in a mouse model of human spinal and bulbar muscular atrophy: implications for therapyMetformin treatment has no beneficial effect in a dose-response survival study in the SOD1(G93A) mouse model of ALS and is harmful in female micePlasma neurofilament heavy chain levels correlate to markers of late stage disease progression and treatment response in SOD1(G93A) mice that model ALSA novel SOD1-ALS mutation separates central and peripheral effects of mutant SOD1 toxicityA conditioning lesion provides selective protection in a rat model of Amyotrophic Lateral Sclerosis.The human G93A-SOD1 mutation in a pre-symptomatic rat model of amyotrophic lateral sclerosis increases the vulnerability to a mild spinal cord compressionPeripheral hyperstimulation alters site of disease onset and course in SOD1 ratsPhysiological characterization of muscle strength with variable levels of dystrophin restoration in mdx mice following local antisense therapy.Systemic down-regulation of delta-9 desaturase promotes muscle oxidative metabolism and accelerates muscle function recovery following nerve injury.Spinal cord trauma and the molecular point of no returnEarly vulnerability to ischemia/reperfusion injury in motor terminals innervating fast muscles of SOD1-G93A mice.Deletion of the BH3-only protein puma protects motoneurons from ER stress-induced apoptosis and delays motoneuron loss in ALS miceTime-course and characterization of orolingual motor deficits in B6SJL-Tg(SOD1-G93A)1Gur/J miceA nonsense mutation in mouse Tardbp affects TDP43 alternative splicing activity and causes limb-clasping and body tone defects.A plural role for lipids in motor neuron diseases: energy, signaling and structure.Delayed functional recovery in presymptomatic mSOD1(G93A) mice following facial nerve crush axotomy.SOD1-G93A mice exhibit muscle-fiber-type-specific decreases in glucose uptake in the absence of whole-body changes in metabolismThe Role of Skeletal Muscle in Amyotrophic Lateral Sclerosis.Amyotrophic lateral sclerosis and denervation alter sphingolipids and up-regulate glucosylceramide synthasePresymptomatic biochemical changes in hindlimb muscle of G93A human Cu/Zn superoxide dismutase 1 transgenic mouse model of amyotrophic lateral sclerosisWhat is "Hyper" in the ALS Hypermetabolism?Treatment with a coinducer of the heat shock response delays muscle denervation in the SOD1-G93A mouse model of amyotrophic lateral sclerosis
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P2860
The effect of peripheral nerve injury on disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
The effect of peripheral nerve injury on disease progression in the SOD1
@nl
The effect of peripheral nerve ...... amyotrophic lateral sclerosis.
@en
type
label
The effect of peripheral nerve injury on disease progression in the SOD1
@nl
The effect of peripheral nerve ...... amyotrophic lateral sclerosis.
@en
prefLabel
The effect of peripheral nerve injury on disease progression in the SOD1
@nl
The effect of peripheral nerve ...... amyotrophic lateral sclerosis.
@en
P2093
P1433
P1476
The effect of peripheral nerve ...... amyotrophic lateral sclerosis.
@en
P2093
J R T Dick
L Greensmith
P304
P356
10.1016/J.NEUROSCIENCE.2004.09.069
P407
P577
2005-01-01T00:00:00Z