about
Comparative analysis of hepatitis C virus phylogenies from coding and non-coding regions: the 5' untranslated region (UTR) fails to classify subtypesTransmission of single HIV-1 genomes and dynamics of early immune escape revealed by ultra-deep sequencing.Mosaic vaccines elicit CD8+ T lymphocyte responses that confer enhanced immune coverage of diverse HIV strains in monkeys.Mosaic HIV-1 vaccines expand the breadth and depth of cellular immune responses in rhesus monkeys.Human APOBEC3 induced mutation of human immunodeficiency virus type-1 contributes to adaptation and evolution in natural infection.Evidence from small-subunit ribosomal RNA sequences for a fungal origin of Microsporidia.Designing and testing broadly-protective filoviral vaccines optimized for cytotoxic T-lymphocyte epitope coverageA multiple-alignment based primer design algorithm for genetically highly variable DNA targetsEpitope-specific CD8+ T lymphocytes cross-recognize mutant simian immunodeficiency virus (SIV) sequences but fail to contain very early evolution and eventual fixation of epitope escape mutations during SIV infection.Distinct evolutionary pressures underlie diversity in simian immunodeficiency virus and human immunodeficiency virus lineages.Integrated sequence and immunology filovirus database at Los AlamosEffective Cytotoxic T Lymphocyte Targeting of Persistent HIV-1 during Antiretroviral Therapy Requires Priming of Naive CD8+ T CellsExpanded breadth of the T-cell response to mosaic human immunodeficiency virus type 1 envelope DNA vaccinationErratum for Smith et al., Effective Cytotoxic T Lymphocyte Targeting of Persistent HIV-1 during Antiretroviral Therapy Requires Priming of Naive CD8+ T Cells.Immunological and virological mechanisms of vaccine-mediated protection against SIV and HIV.Rare HIV-1 transmitted/founder lineages identified by deep viral sequencing contribute to rapid shifts in dominant quasispecies during acute and early infectionProtection against a mixed SHIV challenge by a broadly neutralizing antibody cocktail.Correction: Rare HIV-1 transmitted/founder lineages identified by deep viral sequencing contribute to rapid shifts in dominant quasispecies during acute and early infection.Molecular evidence for the early evolution of photosynthesis.Coping with viral diversity in HIV vaccine design: a response to Nickle et al.Genotype 1 and global hepatitis C T-cell vaccines designed to optimize coverage of genetic diversity.Human leukocyte antigen-associated sequence polymorphisms in hepatitis C virus reveal reproducible immune responses and constraints on viral evolution.Increased sequence diversity coverage improves detection of HIV-specific T cell responses.P20-21 LB. Gene-to-gene differences in evolutionary rate between HIV-1 and natural SIV from sooty mangabeys: implications for vaccine tests in non-human primates.Web-based design and evaluation of T-cell vaccine candidates.Polyvalent vaccines for optimal coverage of potential T-cell epitopes in global HIV-1 variants.Structural Transition and Antibody Binding of EBOV GP and ZIKV E Proteins from Pre-Fusion to Fusion-Initiation State.P16-13. A greater breadth of HIV-1-specific T cell responses detected using mosaic peptides compare to consensus peptides.P09-21 LB. Deep sequencing of HIV-1 from acute infection: low initial diversity, and rapid but variable CTL escapeSparse coding of pathology slides compared to transfer learning with deep neural networksT cell-based strategies for HIV-1 vaccinesSpike mutation pipeline reveals the emergence of a more transmissible form of SARS-CoV-2Vaccine Advances against Venezuelan, Eastern, and Western Equine Encephalitis Viruses
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Will Fischer
@ast
Will Fischer
@en
Will Fischer
@es
Will Fischer
@nl
Will Fischer
@sl
type
label
Will Fischer
@ast
Will Fischer
@en
Will Fischer
@es
Will Fischer
@nl
Will Fischer
@sl
prefLabel
Will Fischer
@ast
Will Fischer
@en
Will Fischer
@es
Will Fischer
@nl
Will Fischer
@sl
P1053
B-1323-2013
P106
P1153
15759657800
P2456
P31
P3829
P496
0000-0003-4579-4062