Apamin-Sensitive K+ Current Upregulation in Volume-Overload Heart Failure is Associated with the Decreased Interaction of CK2 with SK2.
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Patients with Dilated Cardiomyopathy and Sustained Monomorphic Ventricular Tachycardia Show Up-Regulation of KCNN3 and KCNJ2 Genes and CACNG8-Linked Left Ventricular DysfunctionSK channel enhancers attenuate Ca2+-dependent arrhythmia in hypertrophic hearts by regulating mito-ROS-dependent oxidation and activity of RyR.Altered Mitochondrial Metabolism and Mechanosensation in the Failing Heart: Focus on Intracellular Calcium Signaling.Mitochondrial Mechanosensor Microdomains in Cardiovascular Disorders.Pathogenesis of cardiac ischemia reperfusion injury is associated with CK2α-disturbed mitochondrial homeostasis via suppression of FUNDC1-related mitophagy.Pharmacological blockade of small conductance Ca2+-activated K+ channels by ICA reduces arrhythmic load in rats with acute myocardial infarction
P2860
Apamin-Sensitive K+ Current Upregulation in Volume-Overload Heart Failure is Associated with the Decreased Interaction of CK2 with SK2.
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Apamin-Sensitive K+ Current Up ...... d Interaction of CK2 with SK2.
@en
Apamin-Sensitive K+ Current Up ...... d Interaction of CK2 with SK2.
@nl
type
label
Apamin-Sensitive K+ Current Up ...... d Interaction of CK2 with SK2.
@en
Apamin-Sensitive K+ Current Up ...... d Interaction of CK2 with SK2.
@nl
prefLabel
Apamin-Sensitive K+ Current Up ...... d Interaction of CK2 with SK2.
@en
Apamin-Sensitive K+ Current Up ...... d Interaction of CK2 with SK2.
@nl
P2093
P2860
P1476
Apamin-Sensitive K+ Current Up ...... d Interaction of CK2 with SK2.
@en
P2093
Bingxue Song
Dandan Yang
Feifei Ning
Peijing Hu
Tingzhong Wang
P2860
P2888
P304
P356
10.1007/S00232-015-9839-0
P577
2015-09-11T00:00:00Z