Cellular and molecular basis of increased ammoniagenesis in potassium deprivation.
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Nutritional assessment in cirrhotic patients with hepatic encephalopathyProtein Kinase C Phosphorylates the System N Glutamine Transporter SN1 (Slc38a3) and Regulates Its Membrane Trafficking and DegradationAmmonia transport in the kidney by Rhesus glycoproteins.Perioperative Care of Patients With Liver Cirrhosis: A Review.Serum lactate level and mortality in metformin-associated lactic acidosis requiring renal replacement therapy: a systematic review of case reports and case seriesRenal ammonia metabolism and transport.Estrogen directly and specifically downregulates NaPi-IIa through the activation of both estrogen receptor isoforms (ERα and ERβ) in rat kidney proximal tubule.Mathematical Model of Ammonia Handling in the Rat Renal MedullaEffects of K+-deficient diets with and without NaCl supplementation on Na+, K+, and H2O transporters' abundance along the nephron.Renal ammonia excretion in response to hypokalemia: effect of collecting duct-specific Rh C glycoprotein deletion.Intercalated cell-specific Rh B glycoprotein deletion diminishes renal ammonia excretion response to hypokalemia.Proximal tubule-specific glutamine synthetase deletion alters basal and acidosis-stimulated ammonia metabolism.Expression of glutamine synthetase in the mouse kidney: localization in multiple epithelial cell types and differential regulation by hypokalemia.Mechanisms of the effects of acidosis and hypokalemia on renal ammonia metabolism.Acid-base disturbances in intensive care patients: etiology, pathophysiology and treatment.Evidence for ammonium conductance in a mouse thick ascending limb cell line.Klotho/fibroblast growth factor 23- and PTH-independent estrogen receptor-α-mediated direct downregulation of NaPi-IIa by estrogen in the mouse kidneyRoles of renal ammonia metabolism other than in acid-base homeostasis.Electrolyte and Acid-Base Disturbances in End-Stage Liver Disease: A Physiopathological Approach.Integrated compensatory network is activated in the absence of NCC phosphorylationPotassium deprivation: a systems approach.A gut solution for hepatic encephalopathy.Mechanism of Hyperkalemia-Induced Metabolic Acidosis.
P2860
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P2860
Cellular and molecular basis of increased ammoniagenesis in potassium deprivation.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
Cellular and molecular basis of increased ammoniagenesis in potassium deprivation.
@en
Cellular and molecular basis of increased ammoniagenesis in potassium deprivation.
@nl
type
label
Cellular and molecular basis of increased ammoniagenesis in potassium deprivation.
@en
Cellular and molecular basis of increased ammoniagenesis in potassium deprivation.
@nl
prefLabel
Cellular and molecular basis of increased ammoniagenesis in potassium deprivation.
@en
Cellular and molecular basis of increased ammoniagenesis in potassium deprivation.
@nl
P2093
P2860
P1476
Cellular and molecular basis of increased ammoniagenesis in potassium deprivation.
@en
P2093
Faraaz Siddiqui
Farrukh A Chaudhry
Hassane Amlal
Kamyar Zahedi
Shaikh Abu Hossain
P2860
P304
P356
10.1152/AJPRENAL.00010.2011
P577
2011-07-27T00:00:00Z