Inhibition of phosphodiesterases leads to prevention of the mitochondrial permeability transition pore opening and reperfusion injury in cardiac H9c2 cells.
about
Accelerated recovery of mitochondrial membrane potential by GSK-3β inactivation affords cardiomyocytes protection from oxidant-induced necrosis.Limitations and vulnerabilities of the neonatal cardiovascular system: considerations for anesthetic management.Effects of cyclic nucleotide phosphodiesterases (PDEs) on mitochondrial skeletal muscle functions.Long-term oral resveratrol intake provides nutritional preconditioning against myocardial ischemia/reperfusion injury: involvement of VDAC1 downregulation.
P2860
Inhibition of phosphodiesterases leads to prevention of the mitochondrial permeability transition pore opening and reperfusion injury in cardiac H9c2 cells.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年學術文章
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2011年學術文章
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2011年學術文章
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name
Inhibition of phosphodiesteras ...... injury in cardiac H9c2 cells.
@en
Inhibition of phosphodiesteras ...... injury in cardiac H9c2 cells.
@nl
type
label
Inhibition of phosphodiesteras ...... injury in cardiac H9c2 cells.
@en
Inhibition of phosphodiesteras ...... injury in cardiac H9c2 cells.
@nl
prefLabel
Inhibition of phosphodiesteras ...... injury in cardiac H9c2 cells.
@en
Inhibition of phosphodiesteras ...... injury in cardiac H9c2 cells.
@nl
P2093
P2860
P1476
Inhibition of phosphodiesteras ...... n injury in cardiac H9c2 cells
@en
P2093
David A Zvara
Rachel McIntosh
SungRyul Lee
Xiangjun Shen
Zhelong Xu
P2860
P304
P356
10.1007/S10557-011-6310-Z
P50
P577
2011-08-01T00:00:00Z