Applied Pathology for Interventions of Coronary Chronic Total Occlusion.

about

Applied Pathology for Interventions of Coronary Chronic Total Occlusion.

Item

http://www.wikidata.org/entity/Q54863191

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年学术文章

@wuu

2015年学术文章

@zh-cn

2015年学术文章

@zh-hans

2015年学术文章

@zh-my

2015年学术文章

@zh-sg

2015年學術文章

@yue

2015年學術文章

@zh

2015年學術文章

@zh-hant

name

Applied Pathology for Interventions of Coronary Chronic Total Occlusion.

@en

Applied Pathology for Interventions of Coronary Chronic Total Occlusion.

@nl

type

Item

label

Applied Pathology for Interventions of Coronary Chronic Total Occlusion.

@en

Applied Pathology for Interventions of Coronary Chronic Total Occlusion.

@nl

prefLabel

Applied Pathology for Interventions of Coronary Chronic Total Occlusion.

@en

Applied Pathology for Interventions of Coronary Chronic Total Occlusion.

@nl

P1476

Vascular Calcification Revisited: A New Perspective for Phosphate Transport

@en

P2093

Ricardo Villa-Bellosta

http://www.w3.org/2001/XMLSchema#string

P2860

Role of the mouse ank gene in control of tissue calcification and arthritis

Heterozygous mutations in ANKH, the human ortholog of the mouse progressive ankylosis gene, result in craniometaphyseal dysplasia

SLC34A3 mutations in patients with hereditary hypophosphatemic rickets with hypercalciuria predict a key role for the sodium-phosphate cotransporter NaPi-IIc in maintaining phosphate homeostasis

CFTR regulates outwardly rectifying chloride channels through an autocrine mechanism involving ATP

Kinetic analysis of substrate utilization by native and TNAP-, NPP1-, or PHOSPHO1-deficient matrix vesicles

Autosomal dominant craniometaphyseal dysplasia is caused by mutations in the transmembrane protein ANK

Hereditary hypophosphatemic rickets with hypercalciuria is caused by mutations in the sodium-phosphate cotransporter gene SLC34A3.

Autosomal dominant familial calcium pyrophosphate dihydrate deposition disease is caused by mutation in the transmembrane protein ANKH

NT5E mutations and arterial calcifications

P304

341-351

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.2174/1573403X11666150805120505

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-11-06T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

26242187

http://www.w3.org/2001/XMLSchema#string

P932

4774640

http://www.w3.org/2001/XMLSchema#string