Sudden Unexplained Nocturnal Death Syndrome: The Hundred Years' Enigma.

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Sudden Unexplained Nocturnal Death Syndrome: The Hundred Years' Enigma.

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http://www.wikidata.org/entity/Q55177920

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2018 nî lūn-bûn

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2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

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Sudden Unexplained Nocturnal Death Syndrome: The Hundred Years' Enigma.

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Sudden Unexplained Nocturnal Death Syndrome: The Hundred Years' Enigma.

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Item

label

Sudden Unexplained Nocturnal Death Syndrome: The Hundred Years' Enigma.

@en

Sudden Unexplained Nocturnal Death Syndrome: The Hundred Years' Enigma.

@nl

prefLabel

Sudden Unexplained Nocturnal Death Syndrome: The Hundred Years' Enigma.

@en

Sudden Unexplained Nocturnal Death Syndrome: The Hundred Years' Enigma.

@nl

P1476

Sudden Unexplained Nocturnal Death Syndrome: The Hundred Years' Enigma.

@en

P2093

Da Zheng

http://www.w3.org/2001/XMLSchema#string

Jianding Cheng

http://www.w3.org/2001/XMLSchema#string

Jingjing Zheng

http://www.w3.org/2001/XMLSchema#string

Terry Su

http://www.w3.org/2001/XMLSchema#string

P2860

Identification of mutations in the cardiac ryanodine receptor gene in families affected with arrhythmogenic right ventricular cardiomyopathy type 2 (ARVD2)

A ubiquitous splice variant and a common polymorphism affect heterologous expression of recombinant human SCN5A heart sodium channels

Role of the intercalated disc in cardiac propagation and arrhythmogenesis

Calcium Channel Mutations in Cardiac Arrhythmia Syndromes

Mutations in the cardiac ryanodine receptor gene (hRyR2) underlie catecholaminergic polymorphic ventricular tachycardia

Congenital sick sinus syndrome caused by recessive mutations in the cardiac sodium channel gene (SCN5A)

Arrhythmogenic right ventricular dysplasia/cardiomyopathy: pathogenic desmosome mutations in index-patients predict outcome of family screening: Dutch arrhythmogenic right ventricular dysplasia/cardiomyopathy genotype-phenotype follow-up study

Cardiac-myocyte-specific excision of the vinculin gene disrupts cellular junctions, causing sudden death or dilated cardiomyopathy

Intercalated disc abnormalities, reduced Na(+) current density, and conduction slowing in desmoglein-2 mutant mice prior to cardiomyopathic changes

Missense mutations in plakophilin-2 cause sodium current deficit and associate with a Brugada syndrome phenotype

P31

scholarly article

P356

10.1161/JAHA.117.007837

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2018-03-03T00:00:00Z

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P698

29502107

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P932

5866328

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