Reactive oxygen species mediate N-(4-hydroxyphenyl)retinamide-induced cell death in malignant T cells and are inhibited by the HTLV-I oncoprotein Tax.
about
HTLV-1 p13, a small protein with a busy agenda.Dihydroceramide accumulation and reactive oxygen species are distinct and nonessential events in 4-HPR-mediated leukemia cell death.Treatment of adult T-cell leukemia/lymphoma: past, present, and future.HTLV-1 and apoptosis: role in cellular transformation and recent advances in therapeutic approaches.Reduced levels of reactive oxygen species correlate with inhibition of apoptosis, rise in thioredoxin expression and increased bovine leukemia virus proviral loads.Dihydroceramide desaturase activity is modulated by oxidative stress.N-(4-hydroxyphenyl)retinamide promotes apoptosis of resting and proliferating B-cell chronic lymphocytic leukemia cells and potentiates fludarabine and ABT-737 cytotoxicity.
P2860
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P2860
Reactive oxygen species mediate N-(4-hydroxyphenyl)retinamide-induced cell death in malignant T cells and are inhibited by the HTLV-I oncoprotein Tax.
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年學術文章
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name
Reactive oxygen species mediate N-
@nl
Reactive oxygen species mediat ...... by the HTLV-I oncoprotein Tax.
@en
type
label
Reactive oxygen species mediate N-
@nl
Reactive oxygen species mediat ...... by the HTLV-I oncoprotein Tax.
@en
prefLabel
Reactive oxygen species mediate N-
@nl
Reactive oxygen species mediat ...... by the HTLV-I oncoprotein Tax.
@en
P2093
P2860
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Reactive oxygen species mediat ...... by the HTLV-I oncoprotein Tax.
@en
P2093
Abou-Lteif G
Bazarbachi A
Darwiche N
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P2888
P304
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10.1038/SJ.LEU.2404472
P577
2006-11-23T00:00:00Z
P5875
P6179
1048058492