Evidence that UBASH3 is a causal gene for type 1 diabetes.

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Evidence that UBASH3 is a causal gene for type 1 diabetes.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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2018年學術文章

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Evidence that UBASH3 is a causal gene for type 1 diabetes.

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Evidence that UBASH3 is a causal gene for type 1 diabetes.

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label

Evidence that UBASH3 is a causal gene for type 1 diabetes.

@en

Evidence that UBASH3 is a causal gene for type 1 diabetes.

@nl

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Evidence that UBASH3 is a causal gene for type 1 diabetes.

@en

Evidence that UBASH3 is a causal gene for type 1 diabetes.

@nl

P1476

Evidence that UBASH3 is a causal gene for type 1 diabetes.

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P2093

John A Todd

http://www.w3.org/2001/XMLSchema#string

P2860

Regulatory T Cell Responses in Participants with Type 1 Diabetes after a Single Dose of Interleukin-2: A Non-Randomised, Open Label, Adaptive Dose-Finding Trial

Systematic identification of trans eQTLs as putative drivers of known disease associations

Fine mapping of type 1 diabetes susceptibility loci and evidence for colocalization of causal variants with lymphoid gene enhancers.

Pathogenic implications for autoimmune mechanisms derived by comparative eQTL analysis of CD4+ versus CD8+ T cells.

Polarization of the effects of autoimmune and neurodegenerative risk alleles in leukocytes.

Disease variants alter transcription factor levels and methylation of their binding sites.

Identification of context-dependent expression quantitative trait loci in whole blood.

Genetic effects on gene expression across human tissues.

UBASH3A Mediates Risk for Type 1 Diabetes Through Inhibition of T-Cell Receptor-Induced NF-κB Signaling.

Molecular-genetic characterization of common, noncoding UBASH3A variants associated with type 1 diabetes.

P2888

s41431-018-0142-2

P31

scholarly article

P356

10.1038/S41431-018-0142-2

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P577

2018-05-14T00:00:00Z

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P6179

1103970826

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P698

29760431

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P921

type-1 diabetes

P932

6018710

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