about
Activation of H-Ras in the endoplasmic reticulum by the RasGRF family guanine nucleotide exchange factorsThe Borg family of Cdc42 effector proteins Cdc42EP1-5Lysophosphatidic acid rescues RhoA activation and phosphoinositides levels in astrocytes exposed to ethanolRas, an actor on many stages: posttranslational modifications, localization, and site-specified events.TGFβ-mediated suppression of CD248 in non-cancer cells via canonical Smad-dependent signaling pathways is uncoupled in cancer cellsTranscriptomal profiling of site-specific Ras signals.Mesenchymal Cancer Cell-Stroma Crosstalk Promotes Niche Activation, Epithelial Reversion, and Metastatic Colonization.Cdc42EP3/BORG2 and Septin Network Enables Mechano-transduction and the Emergence of Cancer-Associated Fibroblasts.Reactivation of p53 by a Cytoskeletal Sensor to Control the Balance Between DNA Damage and Tumor DisseminationTumour cell-derived Wnt7a recruits and activates fibroblasts to promote tumour aggressiveness.Mechanotransduction and YAP-dependent matrix remodelling is required for the generation and maintenance of cancer-associated fibroblasts.Structural and spatial determinants regulating TC21 activation by RasGRF family nucleotide exchange factorsThe Ras-ERK pathway: understanding site-specific signaling provides hope of new anti-tumor therapies.Cell communication networks in cancer invasion.Ras and Rho GTPases on the move: The RasGRF connection.ERK1/2 MAP kinases promote cell cycle entry by rapid, kinase-independent disruption of retinoblastoma-lamin A complexes.Cdc42 regulates Cdc42EP3 function in cancer-associated fibroblastsRasGRF suppresses Cdc42-mediated tumour cell movement, cytoskeletal dynamics and transformation.Characterisation of HRas local signal transduction networks using engineered site-specific exchange factors.Analysis of Breast Cancer Cell Invasion Using an Organotypic Culture System.Tumor microenvironment: unleashing metalloproteinases to induce a CAF phenotype.ERK1/2 MAP kinases promote cell cycle entry by rapid, kinase-independent disruption of retinoblastoma–lamin A complexes.Regulation of mechanotransduction: Emerging roles for septinsFactors Secreted by Cancer-Associated Fibroblasts that Sustain Cancer Stem Properties in Head and Neck Squamous Carcinoma Cells as Potential Therapeutic TargetsDickkopf-3 links HSF1 and YAP/TAZ signalling to control aggressive behaviours in cancer-associated fibroblastsAn Integrated Global Analysis of Compartmentalized HRAS Signaling
P50
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P50
description
researcher
@en
researcher
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wetenschapper
@nl
հետազոտող
@hy
name
Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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prefLabel
Fernando Calvo
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Fernando Calvo
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Fernando Calvo
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Fernando Calvo
@es
Fernando Calvo
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Fernando Calvo
@sl
P106
P21
P31
P496
0000-0001-8858-1185