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Fibroblast-led collective invasion of carcinoma cells with differing roles for RhoGTPases in leading and following cellsAn open data ecosystem for cell migration research.A chemical biology screen reveals a role for Rab21-mediated control of actomyosin contractility in fibroblast-driven cancer invasion.Diverse matrix metalloproteinase functions regulate cancer amoeboid migrationPluripotent stem cell model reveals essential roles for miR-450b-5p and miR-184 in embryonic corneal lineage specification.Epigenetic switch drives the conversion of fibroblasts into proinvasive cancer-associated fibroblastsKnock out of the BASIGIN/CD147 chaperone of lactate/H+ symporters disproves its pro-tumour action via extracellular matrix metalloproteases (MMPs) induction.Melanoma invasion - current knowledge and future directions.Vascular stiffness mechanoactivates YAP/TAZ-dependent glutaminolysis to drive pulmonary hypertensionp38MAPK builds a hyaluronan cancer niche to drive lung tumorigenesis.Membrane-bound ICAM-1 contributes to the onset of proinvasive tumor stroma by controlling acto-myosin contractility in carcinoma-associated fibroblasts.Interactions of Melanoma Cells with Distal Keratinocytes Trigger Metastasis via Notch Signaling Inhibition of MITF.Fibroblast activation in cancer: when seed fertilizes soil.LIF mediates proinvasive activation of stromal fibroblasts in cancer.Analysis of collective invasion of carcinoma cells in a 3D organotypic model.Myocardin-related transcription factors and SRF are required for cytoskeletal dynamics and experimental metastasis.SPARC represses E-cadherin and induces mesenchymal transition during melanoma development.The cleavage of microphthalmia-associated transcription factor, MITF, by caspases plays an essential role in melanocyte and melanoma cell apoptosisHGF induces fibronectin matrix synthesis in melanoma cells through MAP kinase-dependent signaling pathway and induction of Egr-1.Pathological prion protein exposure switches on neuronal mitogen-activated protein kinase pathway resulting in microglia recruitment.Collective invasion of carcinoma cells: when the fibroblasts take the lead.Hypoxia-inducible factor 1{alpha} is a new target of microphthalmia-associated transcription factor (MITF) in melanoma cells.Microphthalmia-associated transcription factor (MITF) is required but is not sufficient to induce the expression of melanogenic genes.[Carcinoma-associated fibroblasts in cancer: the great escape].Tumor-Stroma Mechanics Coordinate Amino Acid Availability to Sustain Tumor Growth and MalignancyROCK and JAK1 Signaling Cooperate to Control Actomyosin Contractility in Tumor Cells and StromaTumor-Derived Fibronectin Is Involved in Melanoma Cell Invasion and Regulated by V600E B-Raf Signaling PathwayUBTD1 is a mechano-regulator controlling cancer aggressivenessThree-Dimensional Cell Culture Conditions Affect the Proteome of Cancer-Associated FibroblastsMatrix Stiffening and EGFR Cooperate to Promote the Collective Invasion of Cancer CellsA feed-forward mechanosignaling loop confers resistance to therapies targeting the MAPK pathway in BRAF-mutant melanoma
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Cédric Gaggioli
@ast
Cédric Gaggioli
@en
Cédric Gaggioli
@es
Cédric Gaggioli
@nl
type
label
Cédric Gaggioli
@ast
Cédric Gaggioli
@en
Cédric Gaggioli
@es
Cédric Gaggioli
@nl
prefLabel
Cédric Gaggioli
@ast
Cédric Gaggioli
@en
Cédric Gaggioli
@es
Cédric Gaggioli
@nl
P214
P106
P214
P31
P496
0000-0002-4710-1605
P7859
viaf-215006712