about
Transgenic rescue of ataxia mice with neuronal-specific expression of ubiquitin-specific protease 14Mice lacking the transcriptional coactivator PGC-1α exhibit alterations in inhibitory synaptic transmission in the motor cortex.PGC-1α provides a transcriptional framework for synchronous neurotransmitter release from parvalbumin-positive interneurons.Genetic background alters the severity and onset of neuromuscular disease caused by the loss of ubiquitin-specific protease 14 (usp14)Long-term potentiation and the computational synapse.Interneuron Transcriptional Dysregulation Causes Frequency-Dependent Alterations in the Balance of Inhibition and Excitation in Hippocampus.Usp14 deficiency increases tau phosphorylation without altering tau degradation or causing tau-dependent deficits.Coactivation of M(1) muscarinic and alpha1 adrenergic receptors stimulates extracellular signal-regulated protein kinase and induces long-term depression at CA3-CA1 synapses in rat hippocampusCalcium-permeable presynaptic kainate receptors involved in excitatory short-term facilitation onto somatostatin interneurons during natural stimulus patternsDifferential effects of Usp14 and Uch-L1 on the ubiquitin proteasome system and synaptic activity.Endogenously Released Neuropeptide Y Suppresses Hippocampal Short-Term Facilitation and Is Impaired by Stress-Induced Anxiety.Developmental changes in short-term facilitation are opposite at temporoammonic synapses compared to Schaffer collateral synapses onto CA1 pyramidal cells.A catalytic independent function of the deubiquitinating enzyme USP14 regulates hippocampal synaptic short-term plasticity and vesicle number.Novel form of long-term synaptic depression in rat hippocampus induced by activation of alpha 1 adrenergic receptors.Transcriptional dysregulation causes altered modulation of inhibition by haloperidol.Developmental decrease in short-term facilitation at Schaffer collateral synapses in hippocampus is mGluR1 sensitive.Presynaptic kainate receptor activation is a novel mechanism for target cell-specific short-term facilitation at Schaffer collateral synapses.Sympathetic sprouting drives hippocampal cholinergic reinnervation that prevents loss of a muscarinic receptor-dependent long-term depression at CA3-CA1 synapses.Prefrontal cortex-dependent innate behaviors are altered by selective knockdown of Gad1 in neuropeptide Y interneurons.Effect of temperature on Ca(2+)-dependent and mechanical modulators of relaxation in mammalian myocardiumPriming plasticity
P50
Q28271760-4AAD9A4C-1BD7-4D65-B49D-8A0EFBD51EA3Q33799027-1DFDBA9B-CC34-4C9E-8381-98AE623EB5E7Q34383492-DC27C548-C389-4D0C-B5EA-7F30F9D7DEBEQ35073229-3F03A2B7-C913-484B-95E0-F5A0FF9D01C7Q36185407-60DFB1E2-90B3-4C1F-B6EC-0095D5E4D860Q36290045-A22A6205-A560-42CC-9134-D26FDB4F1B6EQ36354014-B9AC48BF-0837-48F8-95C1-A05149A6A7D8Q36763622-83D44714-B49C-45B3-BB54-80E1543DFFB6Q37131763-7ECD15DC-8092-40E6-AA6E-C40A7133E584Q37325929-2358BAD1-C21D-4191-843D-7C3467F1A1B4Q37558885-06DF6F7D-80CD-425B-8719-18C062E7F945Q41878364-E409785D-F83A-45FD-B14C-258BD8AF26EFQ42448520-8A8D8E23-B7CD-4855-A0DF-F01EC91F4A45Q44628469-7251B8DB-55DF-4964-8C14-D9C4E9E102E0Q46349804-9CFC6208-C3E5-4DF9-9DD3-0B388CE631D1Q46886520-27788E45-A2AF-4F3A-A60B-829D4C71C212Q48392815-1BC94EAC-6275-4E3F-826D-E6FAE875A485Q48592346-B257BA38-268B-460D-84BF-85C53F88647DQ64885471-9EBB275F-6AA1-4403-B611-D5599CDBD0A8Q72003219-413446A5-02BA-42DB-802B-A55D7A93DEC8Q77505880-904EDAEA-F3C3-431E-9132-7C7A9CF62B9F
P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Lynn E Dobrunz
@ast
Lynn E Dobrunz
@en
Lynn E Dobrunz
@es
Lynn E Dobrunz
@nl
type
label
Lynn E Dobrunz
@ast
Lynn E Dobrunz
@en
Lynn E Dobrunz
@es
Lynn E Dobrunz
@nl
prefLabel
Lynn E Dobrunz
@ast
Lynn E Dobrunz
@en
Lynn E Dobrunz
@es
Lynn E Dobrunz
@nl
P106
P1153
6601986964
P31
P496
0000-0002-3317-554X