about
Overexpression of Reelin prevents the manifestation of behavioral phenotypes related to schizophrenia and bipolar disorder.Targeting Dyrk1A with AAVshRNA attenuates motor alterations in TgDyrk1A, a mouse model of Down syndrome.DREAM controls the on/off switch of specific activity-dependent transcription pathways.Cognition and hippocampal plasticity in the mouse is altered by monosomy of a genomic region implicated in Down syndromeOpposite phenotypes of muscle strength and locomotor function in mouse models of partial trisomy and monosomy 21 for the proximal Hspa13-App region.Ceramide levels regulated by carnitine palmitoyltransferase 1C control dendritic spine maturation and cognition.Candidate genes for panic disorder: insight from human and mouse genetic studies.A new mouse model for the trisomy of the Abcg1-U2af1 region reveals the complexity of the combinatorial genetic code of down syndromeAn animal model of compulsive food-taking behaviour.Reduced Mid1 Expression and Delayed Neuromotor Development in daDREAM Transgenic Mice.Developmental molecular and functional cerebellar alterations induced by PCP4/PEP19 overexpression: implications for Down syndrome.Carnitine palmitoyltransferase 1C deficiency causes motor impairment and hypoactivity.AGC1-malate aspartate shuttle activity is critical for dopamine handling in the nigrostriatal pathway.Dissociation between CA3-CA1 synaptic plasticity and associative learning in TgNTRK3 transgenic mice.Differential responses to anxiogenic drugs in a mouse model of panic disorder as revealed by Fos immunocytochemistry in specific areas of the fear circuitry.Transgenic mice overexpressing the full-length neurotrophin receptor TrkC exhibit increased catecholaminergic neuron density in specific brain areas and increased anxiety-like behavior and panic reaction.Heterozygous deletion of the Williams-Beuren syndrome critical interval in mice recapitulates most features of the human disorder.Excitation/inhibition balance and learning are modified by Dyrk1a gene dosage.
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description
researcher
@en
wetenschapper
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հետազոտող
@hy
name
Ignasi Sahún
@ast
Ignasi Sahún
@en
Ignasi Sahún
@es
Ignasi Sahún
@nl
type
label
Ignasi Sahún
@ast
Ignasi Sahún
@en
Ignasi Sahún
@es
Ignasi Sahún
@nl
prefLabel
Ignasi Sahún
@ast
Ignasi Sahún
@en
Ignasi Sahún
@es
Ignasi Sahún
@nl
P106
P31
P496
0000-0002-4257-9255