about
Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosisClass II HLA interactions modulate genetic risk for multiple sclerosis.Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis.Gut bacteria from multiple sclerosis patients modulate human T cells and exacerbate symptoms in mouse models.Systematic integration of biomedical knowledge prioritizes drugs for repurposing.The Genetics of Multiple Sclerosis: From 0 to 200 in 50 Years.Integrating biomedical research and electronic health records to create knowledge-based biologically meaningful machine-readable embeddingsGenetic variation across RNA metabolism and cell death gene networks is implicated in the semantic variant of primary progressive aphasiamicroRNA and exosome profiling in multiple sclerosisAberrant oligodendroglial-vascular interactions disrupt the blood-brain barrier, triggering CNS inflammationSerum antibodies to phosphatidylcholine in MSVitamin D Regulates MerTK-Dependent Phagocytosis in Human Myeloid Cells
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Sergio E Baranzini
@ast
Sergio E Baranzini
@en
Sergio E Baranzini
@es
Sergio E Baranzini
@nl
type
label
Sergio E Baranzini
@ast
Sergio E Baranzini
@en
Sergio E Baranzini
@es
Sergio E Baranzini
@nl
prefLabel
Sergio E Baranzini
@ast
Sergio E Baranzini
@en
Sergio E Baranzini
@es
Sergio E Baranzini
@nl
P106
P21
P31
P496
0000-0003-0067-194X