Intron 7 retention and exon 9 skipping EAAT2 mRNA variants are not associated with amyotrophic lateral sclerosis
about
Therapeutic developments in the treatment of amyotrophic lateral sclerosis.Excitotoxicity and amyotrophic lateral sclerosis.Neuronal exosomal miRNA-dependent translational regulation of astroglial glutamate transporter GLT1.Genetic studies of amyotrophic lateral sclerosis: controversies and perspectives.A novel alternative splicing form of excitatory amino acid transporter 1 is a negative regulator of glutamate uptake.The role of glutamate transporters in the pathophysiology of neuropsychiatric disorders.Identification of BRCA1-IRIS, a BRCA1 locus product.
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P2860
Intron 7 retention and exon 9 skipping EAAT2 mRNA variants are not associated with amyotrophic lateral sclerosis
description
article
@en
im Januar 2001 veröffentlichter wissenschaftlicher Artikel
@de
wetenschappelijk artikel
@nl
наукова стаття, опублікована у 2001
@uk
name
Intron 7 retention and exon 9 ...... amyotrophic lateral sclerosis
@en
Intron 7 retention and exon 9 ...... amyotrophic lateral sclerosis
@nl
type
label
Intron 7 retention and exon 9 ...... amyotrophic lateral sclerosis
@en
Intron 7 retention and exon 9 ...... amyotrophic lateral sclerosis
@nl
prefLabel
Intron 7 retention and exon 9 ...... amyotrophic lateral sclerosis
@en
Intron 7 retention and exon 9 ...... amyotrophic lateral sclerosis
@nl
P2093
P356
P1433
P1476
Intron 7 retention and exon 9 ...... amyotrophic lateral sclerosis
@en
P2093
Christopher E. Shaw
Joanna M. Flowers
John F. Powell
P304
P356
10.1002/ANA.1029
P577
2001-01-01T00:00:00Z