CD28 and CTLA4 coordinately regulate airway inflammatory cell recruitment and T-helper cell differentiation after inhaled allergen
about
Transcriptomic analysis of the temporal host response to skin infestation with the ectoparasitic mite Psoroptes ovisA randomized controlled trial to evaluate inhibition of T-cell costimulation in allergen-induced airway inflammation.An enigmatic tail of CD28 signaling.Effects of different up-dosing regimens for hymenoptera venom immunotherapy on serum CTLA-4 and IL-10.Influence of Slc11a1 on the outcome of Salmonella enterica serovar Enteritidis infection in mice is associated with Th polarization.Drug development for asthma.Soluble CD86 protein in serum samples of patients with asthma.Role of T cell co-stimulation in murine models of allergic asthma.Enhancement of soluble CD28 levels in the serum of Graves' diseaseSensitization to cockroach allergen: immune regulation and genetic determinants.A dose-dependent requirement for the proline motif of CD28 in cellular and humoral immunity revealed by a targeted knockin mutant.Co-stimulatory molecules as potential targets for therapeutic intervention in allergic airway disease.Targeted knock-in mice expressing mutations of CD28 reveal an essential pathway for costimulationThe role of costimulatory molecules in allergic disease and asthma.Serum biomarkers in elderly asthma.Cutting edge: A double-mutant knockin of the CD28 YMNM and PYAP motifs reveals a critical role for the YMNM motif in regulation of T cell proliferation and Bcl-xL expression.CTLA4-Ig inhibits allergic airway inflammation by a novel CD28-independent, nitric oxide synthase-dependent mechanismCTLA4Ig inhibits effector T cells through regulatory T cells and TGF-β.Functional polymorphism in CTLA4 gene influences the response to therapy with inhaled corticosteroids in Slovenian children with atopic asthma.Effects of allergen inhalation and oral glucocorticoid on serum soluble CTLA-4 in allergic asthmatics.CD28/CTLA4 double deficient mice demonstrate crucial role for B7 co-stimulation in the induction of allergic lower airways disease.Plasma concentrations of soluble CTLA-4, CD28, CD80 and CD86 costimulatory molecules reflect disease severity of acute asthma in childrenAberrant production of soluble costimulatory molecules CTLA-4, CD28, CD80 and CD86 in patients with systemic lupus erythematosusIncreased expression of plasma and cell surface co-stimulatory molecules CTLA-4, CD28 and CD86 in adult patients with allergic asthmaElevation of plasma soluble T cell costimulatory molecules CTLA-4, CD28 and CD80 in children with allergic asthma
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CD28 and CTLA4 coordinately regulate airway inflammatory cell recruitment and T-helper cell differentiation after inhaled allergen
description
im Mai 2001 veröffentlichter wissenschaftlicher Artikel
@de
wetenschappelijk artikel
@nl
наукова стаття, опублікована в травні 2001
@uk
name
CD28 and CTLA4 coordinately re ...... tiation after inhaled allergen
@en
CD28 and CTLA4 coordinately re ...... tiation after inhaled allergen
@nl
type
label
CD28 and CTLA4 coordinately re ...... tiation after inhaled allergen
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CD28 and CTLA4 coordinately re ...... tiation after inhaled allergen
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prefLabel
CD28 and CTLA4 coordinately re ...... tiation after inhaled allergen
@en
CD28 and CTLA4 coordinately re ...... tiation after inhaled allergen
@nl
P2093
P2860
P356
P1476
CD28 and CTLA4 coordinately re ...... tiation after inhaled allergen
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D R Randolph
S L Kimzey
P2860
P356
10.1165/AJRCMB.24.5.4375
P407
P577
2001-05-01T00:00:00Z