about
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Emerging Roles for the Immune System in Traumatic Brain InjuryDietary modulation of the microbiome affects autoinflammatory diseaseMitochondria: diversity in the regulation of the NLRP3 inflammasome.NLRP6 negatively regulates innate immunity and host defence against bacterial pathogensInfluence of apoA-I and apoE on the formation of serum amyloid A-containing lipoproteins in vivo and in vitroTyrosine Kinase SYK Licenses MyD88 Adaptor Protein to Instigate IL-1α-Mediated Inflammatory Disease.Oxidized Low-Density Lipoprotein Immune Complex Priming of the Nlrp3 Inflammasome Involves TLR and FcγR Cooperation and Is Dependent on CARD9The brain's reward circuitry regulates immunity.Dealing with Danger in the CNS: The Response of the Immune System to Injury.The NLRP12 Sensor Negatively Regulates Autoinflammatory Disease by Modulating Interleukin-4 Production in T Cells.Drak2 is not required for tumor surveillance and suppressionMHCII-independent CD4+ T cells protect injured CNS neurons via IL-4.Blockade of PD-1/B7-H1 interaction restores effector CD8+ T cell responses in a hepatitis C virus core murine modelIntrahepatic IL-10 maintains NKG2A+Ly49- liver NK cells in a functionally hyporesponsive state.Increased Fas ligand expression of CD4+ T cells by HCV core induces T cell-dependent hepatic inflammation.Liver is able to activate naïve CD8+ T cells with dysfunctional anti-viral activity in the murine system.Fas ligand is responsible for CXCR3 chemokine induction in CD4+ T cell-dependent liver damage.Cognate memory CD4+ T cells generated with dendritic cell priming influence the expansion, trafficking, and differentiation of secondary CD8+ T cells and enhance tumor control.SHP-1 and IL-1α conspire to provoke neutrophilic dermatoses.Beyond canonical inflammasomes: emerging pathways in IL-1-mediated autoinflammatory diseaseCutting edge: critical role for PYCARD/ASC in the development of experimental autoimmune encephalomyelitis.4F Peptide reduces nascent atherosclerosis and induces natural antibody production in apolipoprotein E-null mice.Signaling via the RIP2 adaptor protein in central nervous system-infiltrating dendritic cells promotes inflammation and autoimmunity.Inflammasome activation in obesity-related inflammatory diseases and autoimmunity.Characterization of the natural killer T-cell response in an adoptive transfer model of atherosclerosis.Serum Amyloid A Facilitates Early Lesion Development in Ldlr-/- MiceIL-1 family cytokines trigger sterile inflammatory disease.RIP1-driven autoinflammation targets IL-1α independently of inflammasomes and RIP3.Inflammasome-derived IL-1β regulates the production of GM-CSF by CD4(+) T cells and γδ T cellsCritical role for inflammasome-independent IL-1β production in osteomyelitis.Apolipoprotein A-I protection against atherosclerosis is dependent on genetic background.Serum paraoxonase: effect of the apolipoprotein composition of HDL and the acute phase response.Genetic background selectively influences innominate artery atherosclerosis: immune system deficiency as a probe.Site-specific influence of polyunsaturated fatty acids on atherosclerosis in immune incompetent LDL receptor deficient mice.Lipid-lowering effects of ethyl 2-phenacyl-3-aryl-1H-pyrrole- 4-carboxylates in rodents.Autoantibodies to OxLDL fail to alter the clearance of injected OxLDL in apolipoprotein E-deficient mice.NLR-Dependent Regulation of Inflammation in Multiple Sclerosis- Dependent Regulation of Inflammation in Multiple Sclerosis.Cutting Edge: Critical Roles for Microbiota-Mediated Regulation of the Immune System in a Prenatal Immune Activation Model of AutismTh17 Cells in Parkinson's Disease: The Bane of the MidbrainMHCII-independent CD4+ T cells protect injured CNS neurons via IL-4
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John R Lukens
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John R Lukens
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John R. Lukens
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J. R. Lukens
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John Lukens
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John R Lukens
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Lukens J. R.
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Lukens J.
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0000-0002-6795-0866