about
Affinity-based proteomics reveal cancer-specific networks coordinated by Hsp90Functional screen of MSI2 interactors identifies an essential role for SYNCRIP in myeloid leukemia stem cells.PRMT4 blocks myeloid differentiation by assembling a methyl-RUNX1-dependent repressor complex.The polycomb group protein L3MBTL1 represses a SMAD5-mediated hematopoietic transcriptional program in human pluripotent stem cells.The leukemogenicity of AML1-ETO is dependent on site-specific lysine acetylation.MSI2 is required for maintaining activated myelodysplastic syndrome stem cells.Post-translational modifications of Runx1 regulate its activity in the cellHistone-modifying enzymes: their role in the pathogenesis of acute leukemia and their therapeutic potential.Patient-derived xenotransplants can recapitulate the genetic driver landscape of acute leukemias.The N6-methyladenosine (m6A)-forming enzyme METTL3 controls myeloid differentiation of normal hematopoietic and leukemia cells.Histone-binding of DPF2 mediates its repressive role in myeloid differentiation.
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description
onderzoeker
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researcher
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հետազոտող
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name
Ly P Vu
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Ly P Vu
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Ly P Vu
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Ly P Vu
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Ly P Vu
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Ly P Vu
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Ly P Vu
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Ly P Vu
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Ly P Vu
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Ly P Vu
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Ly P Vu
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Ly P Vu
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P106
P1153
35206686700
P31
P496
0000-0002-1682-7702