about
Transfer of tolerance to collagen type V suppresses T-helper-cell-17 lymphocyte-mediated acute lung transplant rejectionDirect visualization of protease action on collagen triple helical structure.Therapeutic potential of TGF-β-induced CD4(+) Foxp3(+) regulatory T cells in autoimmune diseases.Induced T regulatory cells suppress osteoclastogenesis and bone erosion in collagen-induced arthritis better than natural T regulatory cells.Synergistic effect of TGF-beta superfamily members on the induction of Foxp3+ TregThe function of BAFF on T helper cells in autoimmunityInflammasome-independent role of apoptosis-associated speck-like protein containing a CARD (ASC) in T cell priming is critical for collagen-induced arthritisIsolation of purified and live Foxp3+ regulatory T cells using FACS sorting on scatter plot.Critical role of all-trans retinoic acid in stabilizing human natural regulatory T cells under inflammatory conditionsProtein kinase D1 is essential for the proinflammatory response induced by hypersensitivity pneumonitis-causing thermophilic actinomycetes Saccharopolyspora rectivirgula.Type V collagen-induced oral tolerance plus low-dose cyclosporine prevents rejection of MHC class I and II incompatible lung allografts.Genomic locus on chromosome 1 regulates susceptibility to spontaneous arthritis in mice deficiency of IL-1RACutting edge: all-trans retinoic acid sustains the stability and function of natural regulatory T cells in an inflammatory milieu.Antigen-specific transforming growth factor β-induced Treg cells, but not natural Treg cells, ameliorate autoimmune arthritis in mice by shifting the Th17/Treg cell balance from Th17 predominance to Treg cell predominance.Adoptive transfer of human gingiva-derived mesenchymal stem cells ameliorates collagen-induced arthritis via suppression of Th1 and Th17 cells and enhancement of regulatory T cell differentiationImmunopathogenesis of collagen arthritis.Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions.Analog peptides of type II collagen can suppress arthritis in HLA-DR4 (DRB1*0401) transgenic mice.An HLA-DR1 transgene confers susceptibility to collagen-induced arthritis elicited with human type II collagen.Comprehensive mass spectrometric mapping of the hydroxylated amino acid residues of the α1(V) collagen chain.Th-17, monokines, collagen type V, and primary graft dysfunction in lung transplantation.6β-Hydroxytestosterone, a Cytochrome P450 1B1-Testosterone-Metabolite, Mediates Angiotensin II-Induced Renal Dysfunction in Male Mice.Monomeric, porous type II collagen scaffolds promote chondrogenic differentiation of human bone marrow mesenchymal stem cells in vitro.Characterization of T cell phenotype and function in a double transgenic (collagen-specific TCR/HLA-DR1) humanized model of arthritis.Porphyromonas gingivalis oral infection exacerbates the development and severity of collagen-induced arthritis.The Role of Leukocyte-Associated Ig-like Receptor-1 in Suppressing Collagen-Induced Arthritis.The CII-specific autoimmune T-cell response develops in the presence of FTY720 but is regulated by enhanced Treg cells that inhibit the development of autoimmune arthritis.Bone loss and aggravated autoimmune arthritis in HLA-DRβ1-bearing humanized mice following oral challenge with Porphyromonas gingivalisA role for Apolipoprotein A-I in the pathogenesis of multiple sclerosis.Cytochrome P450 1B1 Contributes to the Development of Angiotensin II-Induced Aortic Aneurysm in Male Apoe(-/-) Mice.T cells stimulated with an analog peptide of type II collagen require the Fc receptor γ-chain to secrete interleukin-4 and suppress autoimmune arthritis in mice.Peptide ligand structure and I-Aq binding avidity influence T cell signaling pathway utilization.Modulation of collagen-induced arthritis by adenovirus-mediated intra-articular expression of modified collagen type II.TGF-β-induced CD4+Foxp3+ T cells attenuate acute graft-versus-host disease by suppressing expansion and killing of effector CD8+ cells.An autoantigen-specific, highly restricted T cell repertoire infiltrates the arthritic joints of mice in an HLA-DR1 humanized mouse model of autoimmune arthritis.Pathogenesis of collagen-induced arthritis: modulation of disease by arthritogenic T-cell epitope location.Induced CD4+ forkhead box protein-positive T cells inhibit mast cell function and established contact hypersensitivity through TGF-β1.Characterization of the Syk-Dependent T Cell Signaling Response to an Altered Peptide.In Vivo Attenuation of Antibody-Mediated Acute Renal Allograft Rejection by Ex Vivo TGF-β-Induced CD4+Foxp3+ Regulatory T Cells.In Vivo Dual Fluorescence Imaging to Detect Joint Destruction.
P50
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P50
description
researcher
@en
wetenschapper
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հետազոտող
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name
David D Brand
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David D Brand
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David D Brand
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David D Brand
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type
label
David D Brand
@ast
David D Brand
@en
David D Brand
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David D Brand
@nl
prefLabel
David D Brand
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David D Brand
@en
David D Brand
@es
David D Brand
@nl
P106
P21
P31
P496
0000-0002-9260-7963