Cyclic β-1,2-glucan is a brucella virulence factor required for intracellular survival
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Leishmania promastigotes: building a safe niche within macrophagesWhole-Genome Analyses of Speciation Events in Pathogenic BrucellaeIntruders below the radar: molecular pathogenesis of Bartonella sppHow rhizobial symbionts invade plants: the Sinorhizobium-Medicago modelEstablishment of Chronic Infection: Brucella's Stealth StrategyType IV secretion system of Brucella spp. and its effectorsProteomic analysis of detergent resistant membrane domains during early interaction of macrophages with rough and smooth Brucella melitensisInteraction network and localization of Brucella abortus membrane proteins involved in the synthesis, transport, and succinylation of cyclic β-1,2-glucans.Contrasting Lifestyles Within the Host CellProteomic characterization of phagosomal membrane microdomains during phagolysosome biogenesis and evolutionPresence of Bacterial Virulence Gene Homologues in the dibenzo-p-dioxins degrading bacterium Sphingomonas wittichiiAdvancement of knowledge of Brucella over the past 50 years.Brucella cyclic β-1,2-glucan plays a critical role in the induction of splenomegaly in mice.Brucella abortus uses a stealthy strategy to avoid activation of the innate immune system during the onset of infection.Identification and isolation of Brucella suis virulence genes involved in resistance to the human innate immune system.Brucella control of dendritic cell maturation is dependent on the TIR-containing protein Btp1.Genome sequence of Brucella abortus vaccine strain S19 compared to virulent strains yields candidate virulence genesGenome degradation in Brucella ovis corresponds with narrowing of its host range and tissue tropism.The differential interaction of Brucella and ochrobactrum with innate immunity reveals traits related to the evolution of stealthy pathogensThe Leishmania donovani lipophosphoglycan excludes the vesicular proton-ATPase from phagosomes by impairing the recruitment of synaptotagmin V.Brucella melitensis VjbR and C12-HSL regulons: contributions of the N-dodecanoyl homoserine lactone signaling molecule and LuxR homologue VjbR to gene expression.Innate immune recognition of flagellin limits systemic persistence of BrucellaPhosphoglucomutase of Yersinia pestis is required for autoaggregation and polymyxin B resistance.A comprehensive proteogenomic study of the human Brucella vaccine strain 104 M.Thioredoxin 80-activated-monocytes (TAMs) inhibit the replication of intracellular pathogens.Mechanisms of microbial escape from phagocyte killing.Importance of Lipopolysaccharide and Cyclic β-1,2-Glucans in Brucella-Mammalian Infections.Brucella β 1,2 cyclic glucan is an activator of human and mouse dendritic cells.Blocking the expression of syntaxin 4 interferes with initial phagocytosis of Brucella melitensis in macrophagesThe Brucella abortus cyclic beta-1,2-glucan virulence factor is substituted with O-ester-linked succinyl residues.1,2-β-Oligoglucan phosphorylase from Listeria innocuaA B lymphocyte mitogen is a Brucella abortus virulence factor required for persistent infection.Complete genome-wide screening and subtractive genomic approach revealed new virulence factors, potential drug targets against bio-war pathogen Brucella melitensis 16M.Replication of Brucella abortus and Brucella melitensis in fibroblasts does not require Atg5-dependent macroautophagy.Pathogenesis and immunobiology of brucellosis: review of Brucella-host interactionsSelective subversion of autophagy complexes facilitates completion of the Brucella intracellular cycle.Functional and Structural Analysis of a β-Glucosidase Involved in β-1,2-Glucan Metabolism in Listeria innocuaQuorum-sensing and BvrR/BvrS regulation, the type IV secretion system, cyclic glucans, and BacA in the virulence of Brucella ovis: similarities to and differences from smooth brucellaeEvaluation of the Role of the opgGH Operon in Yersinia pseudotuberculosis and Its Deletion during the Emergence of Yersinia pestisDectin-1 and NOD2 mediate cathepsin activation in zymosan-induced arthritis in mice
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Cyclic β-1,2-glucan is a brucella virulence factor required for intracellular survival
description
im Mai 2005 veröffentlichter wissenschaftlicher Artikel
@de
scientific article published on 08 May 2005
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в травні 2005
@uk
name
Cyclic β-1,2-glucan is a brucella virulence factor required for intracellular survival
@en
Cyclic β-1,2-glucan is a brucella virulence factor required for intracellular survival
@nl
type
label
Cyclic β-1,2-glucan is a brucella virulence factor required for intracellular survival
@en
Cyclic β-1,2-glucan is a brucella virulence factor required for intracellular survival
@nl
prefLabel
Cyclic β-1,2-glucan is a brucella virulence factor required for intracellular survival
@en
Cyclic β-1,2-glucan is a brucella virulence factor required for intracellular survival
@nl
P2093
P2860
P50
P356
P1433
P1476
Cyclic beta-1,2-glucan is a Brucella virulence factor required for intracellular survival
@en
P2093
Andrés Eduardo Ciocchini
Beatriz Arellano-Reynoso
Edgardo Moreno
Rodolfo Ugalde
P2860
P2888
P304
P356
10.1038/NI1202
P407
P577
2005-05-08T00:00:00Z
P6179
1037650801