about
Activated human T cells express alternative mRNA transcripts encoding a secreted form of RANKLUnraveling macrophage contributions to bone repairOsteoclasts control reactivation of dormant myeloma cells by remodelling the endosteal niche.Early anti-inflammatory intervention ameliorates axial disease in the proteoglycan-induced spondylitis mouse model of ankylosing spondylitis.Expression profiling in spondyloarthropathy synovial biopsies highlights changes in expression of inflammatory genes in conjunction with tissue remodelling genes.Intrauterine Bone Marrow Transplantation in Osteogenesis Imperfecta Mice Yields Donor Osteoclasts and Osteomacs but Not Osteoblasts.Critical roles for interleukin 1 and tumor necrosis factor alpha in antibody-induced arthritisRegulation of bone biology by prostaglandin endoperoxide H synthases (PGHS): a rose by any other name...Inflammation-driven bone formation in a mouse model of ankylosing spondylitis: sequential not parallel processes.Excessive bone formation in a mouse model of ankylosing spondylitis is associated with decreases in Wnt pathway inhibitorsMacrophages: Their Emerging Roles in Bone.Osteal macrophages: a new twist on coupling during bone dynamics.The novel CXCR4 antagonist POL5551 mobilizes hematopoietic stem and progenitor cells with greater efficiency than Plerixafor.Unravelling the pluripotency paradox in fetal and placental mesenchymal stem cells: Oct-4 expression and the case of The Emperor's New Clothes.Microphthalmia transcription factor regulates the expression of the novel osteoclast factor GPNMB.Responses to the proinflammatory cytokines interleukin-1 and tumor necrosis factor alpha in cells derived from rheumatoid synovium and other joint tissues involve nuclear factor kappaB-mediated induction of the Ets transcription factor ESE-1.CD169(+) macrophages mediate pathological formation of woven bone in skeletal lesions of prostate cancer.Role of bone marrow macrophages in controlling homeostasis and repair in bone and bone marrow niches.An antibody against the colony-stimulating factor 1 receptor depletes the resident subset of monocytes and tissue- and tumor-associated macrophages but does not inhibit inflammation.Soluble lymphotoxin is an important effector molecule in GVHD and GVL.Tissue engineered humanized bone supports human hematopoiesis in vivo.Responses in vivo to purified poly(3-hydroxybutyrate-co-3-hydroxyvalerate) implanted in a murine tibial defect model.Differential transcriptional effects of PTH and estrogen during anabolic bone formation.Osteal macrophages promote in vivo intramembranous bone healing in a mouse tibial injury model.Absence of B cells does not compromise intramembranous bone formation during healing in a tibial injury model.Fracture healing via periosteal callus formation requires macrophages for both initiation and progression of early endochondral ossification.Macrophage-derived oncostatin M contributes to human and mouse neurogenic heterotopic ossifications.Neurological heterotopic ossification following spinal cord injury is triggered by macrophage-mediated inflammation in muscle.Macrophages Driving Heterotopic Ossification: Convergence of Genetically-Driven and Trauma-Driven Mechanisms.CD169(+) macrophages are critical for osteoblast maintenance and promote intramembranous and endochondral ossification during bone repair.Characterization of Normal Murine Carpal Bone Development Prompts Re-Evaluation of Pathologic Osteolysis as the Cause of Human Carpal-Tarsal Osteolysis Disorders.Resting and injury-induced inflamed periosteum contain multiple macrophage subsets that are located at sites of bone growth and regeneration.Hematopoietic stem cell mobilizing agents G-CSF, cyclophosphamide or AMD3100 have distinct mechanisms of action on bone marrow HSC niches and bone formation.β-glucan triggers spondylarthritis and Crohn's disease-like ileitis in SKG miceInhibition of JAK1/2 Tyrosine Kinases Reduces Neurogenic Heterotopic Ossification After Spinal Cord InjuryAntigen-specific suppression of established arthritis in mice by dendritic cells deficient in NF-kappaBRANKL protein is expressed at the pannus-bone interface at sites of articular bone erosion in rheumatoid arthritisMobilization with granulocyte colony-stimulating factor blocks medullar erythropoiesis by depleting F4/80(+)VCAM1(+)CD169(+)ER-HR3(+)Ly6G(+) erythroid island macrophages in the mouseSelf-repopulating recipient bone marrow resident macrophages promote long-term hematopoietic stem cell engraftment
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
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name
Allison R Pettit
@ast
Allison R Pettit
@en
Allison R Pettit
@es
Allison R Pettit
@nl
type
label
Allison R Pettit
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Allison R Pettit
@en
Allison R Pettit
@es
Allison R Pettit
@nl
prefLabel
Allison R Pettit
@ast
Allison R Pettit
@en
Allison R Pettit
@es
Allison R Pettit
@nl
P106
P31
P496
0000-0003-4707-7892