Spinal glial activation and cytokine expression after lumbar root injury in the rat
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Kinematic and dynamic gait compensations in a rat model of lumbar radiculopathy and the effects of tumor necrosis factor-alpha antagonismCapnellene, a natural marine compound derived from soft coral, attenuates chronic constriction injury-induced neuropathic pain in ratsBrief, low frequency stimulation of rat peripheral C-fibres evokes prolonged microglial-induced central sensitization in adults but not in neonatesTransient cervical nerve root compression modulates pain: load thresholds for allodynia and sustained changes in spinal neuropeptide expression.MKK3, an upstream activator of p38, contributes to formalin phase 2 and late allodynia in miceMicroinjection of IL-1β into the trigeminal transition zone produces bilateral NMDA receptor-dependent orofacial hyperalgesia involving descending circuitry.Peripheral formalin injection induces unique spinal cord microglial phenotypic changes.Systemic minocycline differentially influences changes in spinal microglial markers following formalin-induced nociceptionPuerarin alleviates neuropathic pain by inhibiting neuroinflammation in spinal cord.The organizational and activational effects of sex hormones on tactile and thermal hypersensitivity following lumbar nerve root injury in male and female ratsThe analgesic effect on neuropathic pain of retrogradely transported botulinum neurotoxin A involves Schwann cells and astrocytesThe response of spinal microglia to chemotherapy-evoked painful peripheral neuropathies is distinct from that evoked by traumatic nerve injuries.Glial TNFα in the spinal cord regulates neuropathic pain induced by HIV gp120 application in ratsEstrogen alters baseline and inflammatory-induced cytokine levels independent from hypothalamic-pituitary-adrenal axis activityTNFα is involved in neuropathic pain induced by nucleoside reverse transcriptase inhibitor in rats.The potential for salmon fibrin and thrombin to mitigate pain subsequent to cervical nerve root injury.How can animal models inform on the transition to chronic symptoms in whiplash?Regulation of Neurotrophin-3 and Interleukin-1β and Inhibition of Spinal Glial Activation Contribute to the Analgesic Effect of Electroacupuncture in Chronic Neuropathic Pain States of Rats.Repetitive Treatment with Diluted Bee Venom Attenuates the Induction of Below-Level Neuropathic Pain Behaviors in a Rat Spinal Cord Injury ModelHerpes simplex virus vector-mediated expression of interleukin-10 reduces below-level central neuropathic pain after spinal cord injury.A shed form of LDL receptor-related protein-1 regulates peripheral nerve injury and neuropathic pain in rodents.Chemical and mechanical nerve root insults induce differential behavioral sensitivity and glial activation that are enhanced in combination.Dissociation of spinal microglia morphological activation and peripheral inflammation in inflammatory pain models.Ultrastructure of junction areas between neurons and astrocytes in rat supraoptic nuclei.Spinal astrocyte gap junctions contribute to oxaliplatin-induced mechanical hypersensitivityEnhancement by interleukin-1β of AMPA and NMDA receptor-mediated currents in adult rat spinal superficial dorsal horn neurons.Different peripheral tissue injury induces differential phenotypic changes of spinal activated microglia.Dorsal root compression produces myelinated axonal degeneration near the biomechanical thresholds for mechanical behavioral hypersensitivityCytokine mRNA expression in painful radiculopathy.Go-sha-jinki-Gan (GJG) ameliorates allodynia in chronic constriction injury-model mice via suppression of TNF-α expression in the spinal cord.P2 receptors and chronic painModification of neuropathic pain sensation through microglial ATP receptors.At-level neuropathic pain is induced by lumbosacral ventral root avulsion injury and ameliorated by root reimplantation into the spinal cord.Role of interleukin-1beta during pain and inflammation.Toll-like receptor signaling adapter proteins govern spread of neuropathic pain and recovery following nerve injury in male mice.Calcium channel α2δ1 proteins mediate trigeminal neuropathic pain states associated with aberrant excitatory synaptogenesis.Increased interleukin-1α and prostaglandin E2 expression in the spinal cord at 1 day after painful facet joint injury: evidence of early spinal inflammation.A genetically engineered thermally responsive sustained release curcumin depot to treat neuroinflammation.Neuronal and microglial mechanisms of neuropathic painSignaling mechanisms in mirror image pain pathogenesis.
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P2860
Spinal glial activation and cytokine expression after lumbar root injury in the rat
description
im Mai 2000 veröffentlichter wissenschaftlicher Artikel
@de
scientific article published on 01 May 2000
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в травні 2000
@uk
name
Spinal glial activation and cytokine expression after lumbar root injury in the rat
@en
Spinal glial activation and cytokine expression after lumbar root injury in the rat
@nl
type
label
Spinal glial activation and cytokine expression after lumbar root injury in the rat
@en
Spinal glial activation and cytokine expression after lumbar root injury in the rat
@nl
prefLabel
Spinal glial activation and cytokine expression after lumbar root injury in the rat
@en
Spinal glial activation and cytokine expression after lumbar root injury in the rat
@nl
P2093
P1433
P1476
Spinal glial activation and cytokine expression after lumbar root injury in the rat
@en
P2093
J N Weinstein
R W Colburn
P304
P356
10.1097/00007632-200005150-00003
P407
P577
2000-05-01T00:00:00Z