about
Myosin vb is associated with plasma membrane recycling systemsIdentification of proteins in the postsynaptic density fraction by mass spectrometryCultured melanocytes from dilute mutant mice exhibit dendritic morphology and altered melanosome distributionMyosin Va associates with mRNA in ribonucleoprotein particles present in myelinated peripheral axons and in the central nervous systemIntegrative structural modelling of the cardiac thin filament: energetics at the interface and conservation patterns reveal a spotlight on period 2 of tropomyosinMyosin-Va-dependent cell-to-cell transfer of RNA from Schwann cells to axons.Fast adaptation in vestibular hair cells requires myosin-1c activity.Myosin-I nomenclature.Myosin-Vb functions as a dynamic tether for peripheral endocytic compartments during transferrin trafficking.Class V myosins.Myosin-V: head to tail.Mechanistic heterogeneity in contractile properties of α-tropomyosin (TPM1) mutants associated with inherited cardiomyopathies.Chemical-genetic inhibition of a sensitized mutant myosin Vb demonstrates a role in peripheral-pericentriolar membrane traffic.Pathogenesis of acute murine cytomegalovirus infection in resistant and susceptible strains of mice.Pathogenesis of murine cytomegalovirus infection: identification of infected cells in the spleen during acute and latent infections.A retroviral insertion in the dilute (d) locus provides molecular access to this region of mouse chromosome 9.Molecular cloning of a mouse myosin I expressed in brain.Myosin-Va mediates RNA distribution in primary fibroblasts from multiple organs.Transcription in mouse embryo cells permissively infected by murine cytomegalovirus.A chemical-genetic strategy implicates myosin-1c in adaptation by hair cells.Chemical-genetic inhibition of sensitized mutant unconventional myosins.Molecular mechanisms and structural features of cardiomyopathy-causing troponin T mutants in the tropomyosin overlap region.Absence of the tight junctional protein AF-6 disrupts epithelial cell-cell junctions and cell polarity during mouse development.Developmental regulation of ovarian-specific Mos expression.Molecular cloning and restriction endonuclease mapping of the murine cytomegalovirus genome (Smith strain)An interspecific linkage map of mouse chromosome 15 positioned with respect to the centromerePatterns of Evi-1 expression in embryonic and adult tissues suggest that Evi-1 plays an important regulatory role in mouse developmentA molecular genetic linkage map of mouse chromosome 2Selective gene expression in failing human heart. Quantification of steady-state levels of messenger RNA in endomyocardial biopsies using the polymerase chain reactionChromosomal location of three unconventional myosin heavy chain genes in the mouseEngineering of the myosin-ibeta nucleotide-binding pocket to create selective sensitivity to N(6)-modified ADP analogs
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
John A Mercer
@ast
John A Mercer
@en
John A Mercer
@es
John A Mercer
@nl
type
label
John A Mercer
@ast
John A Mercer
@en
John A Mercer
@es
John A Mercer
@nl
prefLabel
John A Mercer
@ast
John A Mercer
@en
John A Mercer
@es
John A Mercer
@nl
P108
P106
P108
P31
P496
0000-0003-0307-4438