Host-Derived TGFB1 Deficiency Suppresses Lesion Development in a Mouse Model of Endometriosis
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Endometriosis: where are we and where are we going?The role of the peritoneum in the pathogenesis of endometriosisTransforming Growth Factor-beta 1 Involved in the Pathogenesis of Endometriosis through Regulating Expression of Vascular Endothelial Growth Factor under Hypoxia.A novel mouse model of endometriosis mimics human phenotype and reveals insights into the inflammatory contribution of shed endometriumRelevant human tissue resources and laboratory models for use in endometriosis research.Possible Loss of GABAergic Inhibition in Mice With Induced Adenomyosis and Treatment With Epigallocatechin-3-Gallate Attenuates the Loss With Improved HyperalgesiaThe peritoneum is both a source and target of TGF-β in women with endometriosis.Transforming growth factor-β induced Warburg-like metabolic reprogramming may underpin the development of peritoneal endometriosis.Involvement of the Wnt/β-catenin signaling pathway in the cellular and molecular mechanisms of fibrosis in endometriosisTGF-βI Regulates Cell Migration through Pluripotent Transcription Factor OCT4 in Endometriosis.Endometriosis, a disease of the macrophagePAI-1 secretion of endometrial and endometriotic cells is Smad2/3- and ERK1/2-dependent and influences cell adhesionPlatelets are an unindicted culprit in the development of endometriosis: clinical and experimental evidence.Non-hormonal targets underlying endometriosis: A focus on molecular mechanisms.ID2 mediates the transforming growth factor-β1-induced Warburg-like effect seen in the peritoneum of women with endometriosis.Platelets impair natural killer cell reactivity and function in endometriosis through multiple mechanisms.Antifibrotic properties of epigallocatechin-3-gallate in endometriosis.Epithelial-to-mesenchymal transition in the development of endometriosis.Ginsenoside Rg3 Decreases Fibrotic and Invasive Nature of Endometriosis by Modulating miRNA-27b: In Vitro and In Vivo Studies.The role of TGF-β in the pathophysiology of peritoneal endometriosis.Endometriosis-Derived Stromal Cells Secrete Thrombin and Thromboxane A2, Inducing Platelet Activation.
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Host-Derived TGFB1 Deficiency Suppresses Lesion Development in a Mouse Model of Endometriosis
description
article
@en
im Dezember 2011 veröffentlichter wissenschaftlicher Artikel
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wetenschappelijk artikel
@nl
наукова стаття, опублікована в березні 2012
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name
Host-Derived TGFB1 Deficiency ...... a Mouse Model of Endometriosis
@en
Host-Derived TGFB1 Deficiency ...... a Mouse Model of Endometriosis
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type
label
Host-Derived TGFB1 Deficiency ...... a Mouse Model of Endometriosis
@en
Host-Derived TGFB1 Deficiency ...... a Mouse Model of Endometriosis
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prefLabel
Host-Derived TGFB1 Deficiency ...... a Mouse Model of Endometriosis
@en
Host-Derived TGFB1 Deficiency ...... a Mouse Model of Endometriosis
@nl
P2093
P1476
Host-derived TGFB1 deficiency ...... a mouse model of endometriosis
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M Louise Hull
M Zahied Johan
Wendy L Hodge
P304
P356
10.1016/J.AJPATH.2011.11.013
P407
P577
2011-12-30T00:00:00Z