Cardiotrophin-1 Is an Osteoclast-Derived Stimulus of Bone Formation Required for Normal Bone Remodeling
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The putative cannabinoid receptor GPR55 affects osteoclast function in vitro and bone mass in vivoOncostatin M promotes bone formation independently of resorption when signaling through leukemia inhibitory factor receptor in miceImpact of inflammation on the osteoblast in rheumatic diseasesBone cell communication factors and SemaphorinsWhat Is Breast in the Bone?Coupling Signals between the Osteoclast and Osteoblast: How are Messages Transmitted between These Temporary Visitors to the Bone Surface?Ciliary neurotrophic factor inhibits bone formation and plays a sex-specific role in bone growth and remodelingGermline deletion of AMP-activated protein kinase beta subunits reduces bone mass without altering osteoclast differentiation or function.Fibrodysplasia ossificans progressiva-related activated activin-like kinase signaling enhances osteoclast formation during heterotopic ossification in muscle tissues.Prostaglandin E2 acts via bone marrow macrophages to block PTH-stimulated osteoblast differentiation in vitro.Vaginally administered PEGylated LIF antagonist blocked embryo implantation and eliminated non-target effects on bone in mice.Osteoclasts: more than 'bone eaters'.Loss of Cbl-PI3K interaction in mice prevents significant bone loss following ovariectomyA review of osteocyte function and the emerging importance of sclerostinZinc finger protein 467 is a novel regulator of osteoblast and adipocyte commitment.Continuous elevation of PTH increases the number of osteoblasts via both osteoclast-dependent and -independent mechanisms.Potential role of odanacatib in the treatment of osteoporosis.Stimulation of Osteogenesis in Bone Defects Implanted with Biodegradable Hydroxyapatite Composed of Rod-Shaped Particles under Mechanical Unloading.Bone cell interactions through Eph/ephrin: bone modeling, remodeling and associated diseases.Serum Amyloid A3 Secreted by Preosteoclasts Inhibits Parathyroid Hormone-stimulated cAMP Signaling in Murine Osteoblasts.Coupling the activities of bone formation and resorption: a multitude of signals within the basic multicellular unitA bone substitute with high affinity for vitamin D-binding protein--relationship with niche of osteoclasts.Stüve-Wiedemann syndrome: LIFR and associated cytokines in clinical course and etiology.Stüve-Wiedemann syndrome and related bent bone dysplasias.Leukemia inhibitory factor: a paracrine mediator of bone metabolism.Intercellular cross-talk among bone cells: new factors and pathways.The interrelationship between bone and fat: from cellular see-saw to endocrine reciprocity.Talking among ourselves: paracrine control of bone formation within the osteoblast lineage.Implications of osteoblast-osteoclast interactions in the management of osteoporosis by antiresorptive agents denosumab and odanacatib.Treatment of post-menopausal osteoporosis: beyond bisphosphonates.Role of osteoclasts in heterotopic ossification enhanced by fibrodysplasia ossificans progressiva-related activin-like kinase 2 mutation in mice.The multi-faceted role of retinoid X receptor in bone remodeling.A Jak1/2 inhibitor, baricitinib, inhibits osteoclastogenesis by suppressing RANKL expression in osteoblasts in vitroBone corticalization requires local SOCS3 activity and is promoted by androgen action via interleukin-6.Osteoimmunology: The Conceptual Framework Unifying the Immune and Skeletal Systems.Effect of intermittent administration of teriparatide on the mechanical and histological changes in bone grafted with β-tricalcium phosphate using a rabbit bone defect model.Oncostatin M regulates osteogenic differentiation of murine adipose-derived mesenchymal progenitor cells through a PKCdelta-dependent mechanism.Coupling factors and exosomal packaging microRNAs involved in the regulation of bone remodelling.Contrasting roles of leukemia inhibitory factor in murine bone development and remodeling involve region-specific changes in vascularization.Osteoclast-secreted SLIT3 coordinates bone resorption and formation.
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Cardiotrophin-1 Is an Osteoclast-Derived Stimulus of Bone Formation Required for Normal Bone Remodeling
description
article
@en
im Dezember 2008 veröffentlichter wissenschaftlicher Artikel
@de
wetenschappelijk artikel
@nl
наукова стаття, опублікована в грудні 2008
@uk
name
Cardiotrophin-1 Is an Osteocla ...... red for Normal Bone Remodeling
@en
Cardiotrophin-1 Is an Osteocla ...... red for Normal Bone Remodeling
@nl
type
label
Cardiotrophin-1 Is an Osteocla ...... red for Normal Bone Remodeling
@en
Cardiotrophin-1 Is an Osteocla ...... red for Normal Bone Remodeling
@nl
prefLabel
Cardiotrophin-1 Is an Osteocla ...... red for Normal Bone Remodeling
@en
Cardiotrophin-1 Is an Osteocla ...... red for Normal Bone Remodeling
@nl
P2093
P356
P1476
Cardiotrophin-1 is an osteocla ...... red for normal bone remodeling
@en
P2093
Elizabeth H Allan
Emma C Walker
Ingrid J Poulton
Julian M W Quinn
Narelle E McGregor
Sueli Pompolo
T John Martin
P304
P356
10.1359/JBMR.080706
P577
2008-12-01T00:00:00Z