Itraconazole perturbs colorectal cancer dormancy through SUFU-mediated WNT inhibition

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Itraconazole perturbs colorectal cancer dormancy through SUFU-mediated WNT inhibition

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scientific article published on 07 August 2018

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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Itraconazole perturbs colorectal cancer dormancy through SUFU-mediated WNT inhibition

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Itraconazole perturbs colorectal cancer dormancy through SUFU-mediated WNT inhibition

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Itraconazole perturbs colorectal cancer dormancy through SUFU-mediated WNT inhibition

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Itraconazole perturbs colorectal cancer dormancy through SUFU-mediated WNT inhibition

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Itraconazole perturbs colorectal cancer dormancy through SUFU-mediated WNT inhibition

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Itraconazole perturbs colorectal cancer dormancy through SUFU-mediated WNT inhibition

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Itraconazole perturbs colorectal cancer dormancy through SUFU-mediated WNT inhibition

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Semiramis A Popova

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Simon J A Buczacki

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Itraconazole, a commonly used antifungal that inhibits Hedgehog pathway activity and cancer growth

A Big Bang model of human colorectal tumor growth.

The consensus molecular subtypes of colorectal cancer

Dll1+ secretory progenitor cells revert to stem cells upon crypt damage.

Visualization and targeting of LGR5+ human colon cancer stem cells.

A paracrine requirement for hedgehog signalling in cancer.

Intestinal label-retaining cells are secretory precursors expressing Lgr5.

Variable clonal repopulation dynamics influence chemotherapy response in colorectal cancer.

Non-Canonical Hedgehog Signaling Is a Positive Regulator of the WNT Pathway and Is Required for the Survival of Colon Cancer Stem Cells.

Itraconazole targets cell cycle heterogeneity in colorectal cancer.

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e1494950

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scholarly article

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10.1080/23723556.2018.1494950

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English

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Simon Buczacki

Semiramis Popova

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2018-08-07T00:00:00Z

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30250932

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colorectal cancer

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6149862

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