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Structural and Functional Studies on the Interaction of GspC and GspD in the Type II Secretion SystemStructural basis for the activation of cholera toxin by human ARF6-GTPLitR, a new transcriptional activator in Vibrio fischeri, regulates luminescence and symbiotic light organ colonizationAlanine racemase mutants of Burkholderia pseudomallei and Burkholderia mallei and use of alanine racemase as a non-antibiotic-based selectable marker.Characterization of receptor-mediated signal transduction by Escherichia coli type IIa heat-labile enterotoxin in the polarized human intestinal cell line T84.Type II heat-labile enterotoxins from 50 diverse Escherichia coli isolates belong almost exclusively to the LT-IIc family and may be prophage encodedTransfer of the cholera toxin A1 polypeptide from the endoplasmic reticulum to the cytosol is a rapid process facilitated by the endoplasmic reticulum-associated degradation pathwayTargeting of cholera toxin and Escherichia coli heat labile toxin in polarized epithelia: role of COOH-terminal KDELADP-ribosylation factor 6 acts as an allosteric activator for the folded but not disordered cholera toxin A1 polypeptide.A mutational analysis of residues in cholera toxin A1 necessary for interaction with its substrate, the stimulatory G protein Gsα.Microtubule motors power plasma membrane tubulation in clathrin-independent endocytosisIdentification of motifs in cholera toxin A1 polypeptide that are required for its interaction with human ADP-ribosylation factor 6 in a bacterial two-hybrid systemComplete Genome Sequence of Escherichia coli ER1821R, a Laboratory K-12 Derivative Engineered To Be Deficient in All Methylcytosine and Methyladenine Restriction Systems.The chromosomal nature of LT-II enterotoxins solved: a lambdoid prophage encodes both LT-II and one of two novel pertussis-toxin-like toxin family members in type II enterotoxigenic Escherichia coli.Whole-Genome Sequencing Identifies In Vivo Acquisition of a blaCTX-M-27-Carrying IncFII Transmissible Plasmid as the Cause of Ceftriaxone Treatment Failure for an Invasive Salmonella enterica Serovar Typhimurium InfectionVesicular transport is not required for the cytoplasmic pool of cholera toxin to interact with the stimulatory alpha subunit of the heterotrimeric g protein.Use of translational fusion of the MrpH fimbrial adhesin-binding domain with the cholera toxin A2 domain, coexpressed with the cholera toxin B subunit, as an intranasal vaccine to prevent experimental urinary tract infection by Proteus mirabilisMutational analysis of ganglioside GM(1)-binding ability, pentamer formation, and epitopes of cholera toxin B (CTB) subunits and CTB/heat-labile enterotoxin B subunit chimeras.Analysis of structure and function of the B subunit of cholera toxin by the use of site-directed mutagenesis.Erratum for McCollister et al., Whole-Genome Sequencing Identifies In Vivo Acquisition of a blaCTX-M-27-Carrying IncFII Transmissible Plasmid as the Cause of Ceftriaxone Treatment Failure for an Invasive Salmonella enterica Serovar Typhimurium InfecProteolytic activation of cholera toxin and Escherichia coli labile toxin by entry into host epithelial cells. Signal transduction by a protease-resistant toxin variant.A class of mutant CHO cells resistant to cholera toxin rapidly degrades the catalytic polypeptide of cholera toxin and exhibits increased endoplasmic reticulum-associated degradation.Glycolipid Crosslinking Is Required for Cholera Toxin to Partition Into and Stabilize Ordered Domains.Dissection of the fusion machine of SARS-coronavirus.Structural characterization of the SARS-coronavirus spike S fusion protein core.Ectopic Expression of the ydaS and ydaT Genes of the Cryptic Prophage Rac of Escherichia coli K-12 May Be Toxic but Do They Really Encode Toxins?: a Case for Using Genetic Context To Understand Function.Functional expression of the tellurite resistance determinant from the IncHI-2 plasmid pMER610.Restriction pattern and polypeptide homology among plasmid-borne mercury resistance determinants.Genetic and physical analysis of plasmid genes expressing inducible resistance of tellurite in Escherichia coli.Heat-Labile Enterotoxins.Construction and characterization of versatile cloning vectors for efficient delivery of native foreign proteins to the periplasm of Escherichia coli.Cyclic AMP-independent effects of cholera toxin on B cell activation. II. Binding of ganglioside GM1 induces B cell activationIdentification of errors among database sequence entries and comparison of correct amino acid sequences for the heat-labile enterotoxins of Escherichia coli and Vibrio choleraePlasmid multimer status and not DNA topology likely affects luciferase assay reproducibility
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P50
description
onderzoeker
@nl
researcher, ORCID id # 0000-0001-7689-3093
@en
name
Michael G Jobling
@ast
Michael G Jobling
@en
Michael G Jobling
@nl
type
label
Michael G Jobling
@ast
Michael G Jobling
@en
Michael G Jobling
@nl
prefLabel
Michael G Jobling
@ast
Michael G Jobling
@en
Michael G Jobling
@nl
P106
P31
P496
0000-0001-7689-3093