Interaction between opposing modes of phospho-regulation of the proneural proteins Ascl1 and Ngn2

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Interaction between opposing modes of phospho-regulation of the proneural proteins Ascl1 and Ngn2

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scientific article published on 01 October 2018

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wetenschappelijk artikel

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наукова стаття, опублікована в жовтні 2018

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Interaction between opposing m ...... neural proteins Ascl1 and Ngn2

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Interaction between opposing m ...... neural proteins Ascl1 and Ngn2

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Interaction between opposing m ...... neural proteins Ascl1 and Ngn2

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Interaction between opposing m ...... neural proteins Ascl1 and Ngn2

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Interaction between opposing m ...... neural proteins Ascl1 and Ngn2

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Interaction between opposing m ...... neural proteins Ascl1 and Ngn2

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P1476

Interaction between opposing m ...... neural proteins Ascl1 and Ngn2

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Anna Philpott

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Laura J.A. Hardwick

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Proneural genes and the specification of neural cell types

Cell cycle-regulated multi-site phosphorylation of Neurogenin 2 coordinates cell cycling with differentiation during neurogenesis

Ascl1 phospho-status regulates neuronal differentiation in a Xenopus developmental model of neuroblastoma

Multi-site phosphorylation regulates NeuroD4 activity during primary neurogenesis: a conserved mechanism amongst proneural proteins.

Post-translational modification of Ngn2 differentially affects transcription of distinct targets to regulate the balance between progenitor maintenance and differentiation.

Regulation of motor neuron specification by phosphorylation of neurogenin 2

Proneural genes in neocortical development.

Cell cycle regulation of proliferation versus differentiation in the central nervous system.

MyoD phosphorylation on multiple C terminal sites regulates myogenic conversion activity.

The phosphorylation status of Ascl1 is a key determinant of neuronal differentiation and maturation in vivo and in vitro.

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129

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scholarly article

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10.12688/WELLCOMEOPENRES.14848.1

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http://www.w3.org/2001/XMLSchema#string

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Anna Philpott

Laura J A Hardwick

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2018-10-01T00:00:00Z

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30430141

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6206610

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