The Effects of Ginsenoside Compound K Against Epilepsy by Enhancing the γ-Aminobutyric Acid Signaling Pathway

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The Effects of Ginsenoside Compound K Against Epilepsy by Enhancing the γ-Aminobutyric Acid Signaling Pathway

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im Januar 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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name

The Effects of Ginsenoside Com ...... butyric Acid Signaling Pathway

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The Effects of Ginsenoside Com ...... butyric Acid Signaling Pathway

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The Effects of Ginsenoside Com ...... butyric Acid Signaling Pathway

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The Effects of Ginsenoside Com ...... butyric Acid Signaling Pathway

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The Effects of Ginsenoside Com ...... butyric Acid Signaling Pathway

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The Effects of Ginsenoside Com ...... butyric Acid Signaling Pathway

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P1476

The Effects of Ginsenoside Com ...... butyric Acid Signaling Pathway

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Chaopeng Li

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Dejian Jiang

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Dong-Sheng Ouyang

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Guirong Zeng

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Honghao Zhou

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Kai Hu

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Lulu Chen

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Luping Zhou

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Qing Gao

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Siyu Chen

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P2860

Glutamate and GABA imbalance following traumatic brain injury

A variant of KCC2 from patients with febrile seizures impairs neuronal Cl- extrusion and dendritic spine formation

NKCC1 transporter facilitates seizures in the developing brain

Behavioural phenotypes of hypomorphic KCC2-deficient mice

The Epidemiology of Global Epilepsy.

Animal models of epilepsy: use and limitations.

Disrupted dentate granule cell chloride regulation enhances synaptic excitability during development of temporal lobe epilepsy.

Sedative but not anxiolytic properties of benzodiazepines are mediated by the GABA(A) receptor alpha1 subtype.

Regulation of GABA(A) receptor subunit expression by pharmacological agents.

Cation-chloride cotransporters NKCC1 and KCC2 as potential targets for novel antiepileptic and antiepileptogenic treatments.

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1020

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scholarly article

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10.3389/FPHAR.2018.01020

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English

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2018-01-01T00:00:00Z

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30254585

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Farmakologi

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6142013

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