about
ProNGF\NGF imbalance triggers learning and memory deficits, neurodegeneration and spontaneous epileptic-like discharges in transgenic miceDirect in vivo intracellular selection of conformation-sensitive antibody domains targeting Alzheimer's amyloid-beta oligomers.Intranasal "painless" human Nerve Growth Factor [corrected] slows amyloid neurodegeneration and prevents memory deficits in App X PS1 miceThe chemokine CXCL12 mediates the anti-amyloidogenic action of painless human nerve growth factor.A NH2 tau fragment targets neuronal mitochondria at AD synapses: possible implications for neurodegeneration.Conformational targeting of intracellular Aβ oligomers demonstrates their pathological oligomerization inside the endoplasmic reticulum.NH2-truncated human tau induces deregulated mitophagy in neurons by aberrant recruitment of Parkin and UCHL-1: implications in Alzheimer's disease.Diabetic patients and retinal proliferation: an evaluation of the role of vascular endothelial growth factor (VEGF).Intrabodies for protein interference in Alzheimer s disease.Correction: Intranasal “painless” Human Nerve Growth Factors Slows Amyloid Neurodegeneration and Prevents Memory Deficits in App X PS1 Mice.NGF steers microglia toward a neuroprotective phenotype.Activity-Induced Amyloid-β Oligomers Drive Compensatory Synaptic Rearrangements in Brain Circuits Controlling Memory of Presymptomatic Alzheimer's Disease MiceImpaired adult neurogenesis is an early event in Alzheimer's disease neurodegeneration, mediated by intracellular Aβ oligomers
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description
researcher ORCID ID = 0000-0002-4633-364X
@en
wetenschapper
@nl
name
Giovanni Meli
@ast
Giovanni Meli
@en
Giovanni Meli
@es
Giovanni Meli
@nl
type
label
Giovanni Meli
@ast
Giovanni Meli
@en
Giovanni Meli
@es
Giovanni Meli
@nl
prefLabel
Giovanni Meli
@ast
Giovanni Meli
@en
Giovanni Meli
@es
Giovanni Meli
@nl
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P21
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P496
0000-0002-4633-364X