A NH2 tau fragment targets neuronal mitochondria at AD synapses: possible implications for neurodegeneration.
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Tangles, Toxicity, and Tau Secretion in AD - New Approaches to a Vexing ProblemHuman wild-type full-length tau accumulation disrupts mitochondrial dynamics and the functions via increasing mitofusins.Tau accumulation causes mitochondrial distribution deficits in neurons in a mouse model of tauopathy and in human Alzheimer's disease brainMorphological and bioenergetic demands underlying the mitophagy in post-mitotic neurons: the pink-parkin pathwayRoles of tau protein in health and disease.The Intersection of NGF/TrkA Signaling and Amyloid Precursor Protein Processing in Alzheimer's Disease NeuropathologyA mitochondrial etiology of Alzheimer and Parkinson diseaseTau causes synapse loss without disrupting calcium homeostasis in the rTg4510 model of tauopathyCalpain dysregulation in Alzheimer's diseasePre-synaptic C-terminal truncated tau is released from cortical synapses in Alzheimer's diseaseThe Interface between Cytoskeletal Aberrations and Mitochondrial Dysfunction in Alzheimer's Disease and Related DisordersSoluble forms of tau are toxic in Alzheimer's disease.Synaptic alterations in the rTg4510 mouse model of tauopathy.Tau accumulation induces synaptic impairment and memory deficit by calcineurin-mediated inactivation of nuclear CaMKIV/CREB signalingSubacute Changes in Cleavage Processing of Amyloid Precursor Protein and Tau following Penetrating Traumatic Brain InjuryTau accumulation impairs mitophagy via increasing mitochondrial membrane potential and reducing mitochondrial ParkinEvidence for prion-like mechanisms in several neurodegenerative diseases: potential implications for immunotherapyProtein truncation as a common denominator of human neurodegenerative foldopathies.Autophagy modulation for Alzheimer's disease therapy.Expression of Tau Produces Aberrant Plasma Membrane Blebbing in Glial Cells Through RhoA-ROCK-Dependent F-Actin Remodeling.Physical Exercise and Brain Mitochondrial Fitness: The Possible Role Against Alzheimer's Disease.Mitophagy and Alzheimer's Disease: Cellular and Molecular Mechanisms.NH2-truncated human tau induces deregulated mitophagy in neurons by aberrant recruitment of Parkin and UCHL-1: implications in Alzheimer's disease.Extracellular truncated tau causes early presynaptic dysfunction associated with Alzheimer's disease and other tauopathiesMultiple mechanisms of extracellular tau spreading in a non-transgenic tauopathy model.Chronic traumatic encephalopathy-integration of canonical traumatic brain injury secondary injury mechanisms with tau pathology.Shedding light on mitophagy in neurons: what is the evidence for PINK1/Parkin mitophagy in vivo?
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A NH2 tau fragment targets neuronal mitochondria at AD synapses: possible implications for neurodegeneration.
description
2010 nî lūn-bûn
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2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
A NH2 tau fragment targets neu ...... cations for neurodegeneration.
@en
A NH2 tau fragment targets neu ...... cations for neurodegeneration.
@nl
type
label
A NH2 tau fragment targets neu ...... cations for neurodegeneration.
@en
A NH2 tau fragment targets neu ...... cations for neurodegeneration.
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prefLabel
A NH2 tau fragment targets neu ...... cations for neurodegeneration.
@en
A NH2 tau fragment targets neu ...... cations for neurodegeneration.
@nl
P2093
P50
P356
P1476
A NH2 tau fragment targets neu ...... ications for neurodegeneration
@en
P2093
Giuseppe Sancesario
Mariateresa Ciotti
Pietro Calissano
Rossana Bussani
Veronica Corsetti
P304
P356
10.3233/JAD-2010-100120
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P577
2010-01-01T00:00:00Z