about
PI3K p110γ deletion attenuates murine atherosclerosis by reducing macrophage proliferation but not polarization or apoptosis in lesions.Humans and Mice Display Opposing Patterns of "Browning" Gene Expression in Visceral and Subcutaneous White Adipose Tissue DepotsWNT5A-JNK regulation of vascular insulin resistance in human obesity.Adiponectin attenuates abdominal aortic aneurysm formation in hyperlipidemic mice.Clonal hematopoiesis associated with TET2 deficiency accelerates atherosclerosis development in miceAn antiangiogenic isoform of VEGF-A contributes to impaired vascularization in peripheral artery diseaseNoncanonical Wnt signaling promotes obesity-induced adipose tissue inflammation and metabolic dysfunction independent of adipose tissue expansionHeart Failure With Preserved Ejection Fraction Induces Beiging in Adipose Tissue.Secreted Frizzled-related Protein 5 Diminishes Cardiac Inflammation and Protects the Heart from Ischemia/Reperfusion Injury.Telomere biology and cardiovascular disease.Obesity-Induced Changes in Adipose Tissue Microenvironment and Their Impact on Cardiovascular Disease.Endothelial Dysfunction in Human Diabetes Is Mediated by Wnt5a-JNK Signaling.Control of cell proliferation in atherosclerosis: insights from animal models and human studies.Glutaredoxin-1 up-regulation induces soluble vascular endothelial growth factor receptor 1, attenuating post-ischemia limb revascularizationAdipokines: a link between obesity and cardiovascular disease.Genetic deficiency of Wnt5a diminishes disease severity in a murine model of rheumatoid arthritis.Tumor suppressor p27(Kip1) undergoes endolysosomal degradation through its interaction with sorting nexin 6.The good, the bad, and the ugly of interleukin-6 signaling.A role for miR-33 in p53 regulation: New perspectives for hematopoietic stem cell research.Activation of non-canonical WNT signaling in human visceral adipose tissue contributes to local and systemic inflammation.Somatic Mutations and Clonal Hematopoiesis: Unexpected Potential New Drivers of Age-Related Cardiovascular Disease.Defective p27 phosphorylation at serine 10 affects vascular reactivity and increases abdominal aortic aneurysm development via Cox-2 activation.Increased gene dosage of the Ink4/Arf locus does not attenuate atherosclerosis development in hypercholesterolaemic mice.Tet2-Mediated Clonal Hematopoiesis Accelerates Heart Failure Through a Mechanism Involving the IL-1β/NLRP3 Inflammasome.Quantification of Cellular Proliferation in Mouse Atherosclerotic Lesions.Loss of p27 phosphorylation at Ser10 accelerates early atherogenesis by promoting leukocyte recruitment via RhoA/ROCK.TLR4 in Atherogenesis: Paying the Toll for Antimicrobial Defense.Deficient p27 phosphorylation at serine 10 increases macrophage foam cell formation and aggravates atherosclerosis through a proliferation-independent mechanism.Increased p53 gene dosage reduces neointimal thickening induced by mechanical injury but has no effect on native atherosclerosisAnimal models of atherosclerosisPotential Therapeutic Value of Interleukin 1b-targeted Strategies in Atherosclerotic Cardiovascular DiseaseClonal Hematopoiesis of Indeterminate Potential Reshapes Age-Related CVD: JACC Review Topic of the WeekIntegrated Stress Response Inhibition in Atherosclerosis: Preventing the Stressed-Out Plaque
P50
Q31133175-2779AC86-CEC7-4724-9619-98A2BFC6F8C4Q33639090-BCC561F6-93D9-4E90-AB1D-D48A73884C0EQ33853951-A6D7D53B-1F6B-488C-9447-B4E7C4B9A332Q33910404-A79CFFEA-66F3-457E-A0F1-CB70AAB5B6D1Q34549707-4DF08B02-6FFC-49DE-93B8-CBBC5AE3F157Q34641120-F7BA44C3-E9FB-4DED-AB16-E6ABBA31957AQ35220578-2391BD53-8DFE-4D17-B73E-AED6716EE139Q35882942-5B798C9A-68C3-41C6-8154-D39EB047A0AFQ36548318-A308C85D-EA88-4118-843B-440F18D6BF91Q36661337-736DF847-4BA9-4636-90AB-1EF2525C88FBQ36951691-5E1927C1-F1BF-44ED-AFB7-1203B70107A2Q37021140-5316D007-F5CC-457D-98F2-BF53EB167921Q37630073-B582ECAF-0DFE-419C-8DC2-05973A3FA4BBQ37653350-4F6BD941-B10C-4F9C-8A6E-4CB95C308197Q37707281-1EF6C9DC-5F7C-4C66-8631-E4102994B61CQ38671626-A3717D9B-ECEB-43FC-B441-67DEDB7D70B0Q39727871-F0DA7B27-021F-44C0-97A4-84ABFD9F8F6BQ42085795-EC14DB44-F8EA-46EB-ADDF-DD9B71219D7EQ45240883-47C822F1-A5F5-46D7-AB1D-B73F15D29EEAQ47104117-5CD4859A-7648-4769-8F29-94844C4FED80Q50115925-7F8793C8-79BA-4B22-AED9-A9877143B176Q50148829-D227D158-7681-4189-A7C6-3E766573C725Q51369737-E983B9C8-7BEE-4C0F-99D1-ED76CAECBB36Q52730911-DF0AE399-E86D-4CEB-9310-380BD832FE37Q53319121-EC797100-5697-4D05-8533-AC61A8674356Q53531908-3545881C-8453-4BAD-A5C2-E78CB4DEBF6FQ54241013-88EE6754-F518-431A-9A67-2EBDFD3EABDDQ54563792-E2147FA5-D3B4-4842-BB57-F72991B88A03Q57562752-9AD46ABC-211A-4FE7-857E-DFD3F3D3E7D5Q57761322-C46777F7-E218-4CFE-A8A8-27E1F69B4235Q91947338-2BBE86A7-08EC-4914-84D2-0B23E40C3C6FQ92181769-92A837CB-E483-462C-B017-50685EDCE54DQ92365636-6BBE99F6-83F2-45B0-BEE8-A2B3D2B53EBA
P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-5970-629X
@en
name
José J Fuster
@ast
José J Fuster
@en
José J Fuster
@nl
type
label
José J Fuster
@ast
José J Fuster
@en
José J Fuster
@nl
prefLabel
José J Fuster
@ast
José J Fuster
@en
José J Fuster
@nl
P108
P106
P108
P21
P31
P496
0000-0002-5970-629X