about
Age-related decrease of meiotic cohesins in human oocytesScreening of genes involved in chromosome segregation during meiosis I: toward the identification of genes responsible for infertility in humansCharacterization of a novel mouse gene encoding an SYCP3-like protein that relocalizes from the XY body to the nucleolus during prophase of male meiosis IHORMAD2 is essential for synapsis surveillance during meiotic prophase via the recruitment of ATR activityHORMAD1-dependent checkpoint/surveillance mechanism eliminates asynaptic oocytesPolymorphisms of the 22q11.2 breakpoint region influence the frequency of de novo constitutional t(11;22)s in sperm.Paternal origin of the de novo constitutional t(11;22)(q23;q11).Requirement of DLG1 for cardiovascular development and tissue elongation during cochlear, enteric, and skeletal development: possible role in convergent extension.DNA secondary structure is influenced by genetic variation and alters susceptibility to de novo translocation.The constitutional t(11;22): implications for a novel mechanism responsible for gross chromosomal rearrangements.Chromosomal translocations mediated by palindromic DNA.Impaired DNA replication prompts deletions within palindromic sequences, but does not induce translocations in human cells.Recent advance in our understanding of the molecular nature of chromosomal abnormalities.Fatty acid-binding protein 7 regulates function of caveolae in astrocytes through expression of caveolin-1.Palindromic AT-rich repeat in the NF1 gene is hypervariable in humans and evolutionarily conserved in primates.Cell type-specific occurrence of caveolin-1alpha and -1beta in the lung caused by expression of distinct mRNAs.Genetic variation affects de novo translocation frequency.Chromosomal instability mediated by non-B DNA: cruciform conformation and not DNA sequence is responsible for recurrent translocation in humans.Screening of genes involved in chromosome segregation during meiosis I: in vitro gene transfer to mouse fetal oocytes.The DNA Damage Checkpoint Eliminates Mouse Oocytes with Chromosome Synapsis Failure.In situ detection of gonadotropin-releasing hormone (GnRH) receptor mRNA expression in the rat ovarian follicles.Mechanism of complex gross chromosomal rearrangements: a commentary on concomitant microduplications of MECP2 and ATRX in male patients with severe mental retardation.In vitro organogenesis of gut-like structures from mouse embryonic stem cells.Two different forms of palindrome resolution in the human genome: deletion or translocation.Increased levels of pregnancy-associated plasma protein-A2 in the serum of pre-eclamptic patients.Characterization of gonadotropin-releasing hormone (GnRH)-immunoreactive protein in the rat pineal glandCaveolae: from a morphological point of viewEvidence for gonadotropin-releasing hormone receptor mRNA expression by estrogen in rat granulosa cellsOverproduction of the follistatin-related gene protein in the placenta and maternal serum of women with pre-eclampsiaCaveolin-1 in mesangial cells suppresses MAP kinase activation and cell proliferation induced by bFGF and PDGFDirect ovarian effects of a GnRH agonist in hypophysectomized immature rats: inhibition of theca-interstitial luteinizing hormone receptor mRNA expression and induction of interstitial cell differentiationLong-term Pilocarpine Treatment Improves Salivary Flow in Irradiated Mice
P50
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0003-0584-7158
@en
name
Hiroshi Kogo
@ast
Hiroshi Kogo
@en
Hiroshi Kogo
@es
Hiroshi Kogo
@nl
type
label
Hiroshi Kogo
@ast
Hiroshi Kogo
@en
Hiroshi Kogo
@es
Hiroshi Kogo
@nl
prefLabel
Hiroshi Kogo
@ast
Hiroshi Kogo
@en
Hiroshi Kogo
@es
Hiroshi Kogo
@nl
P106
P1153
56490232200
P31
P496
0000-0003-0584-7158