about
Systems mechanobiology: tension-inhibited protein turnover is sufficient to physically control gene circuits.Simple insoluble cues specify stem cell differentiationContractile forces sustain and polarize hematopoiesis from stem and progenitor cells.Matrix elasticity regulates lamin-A,C phosphorylation and turnover with feedback to actomyosin.Stress sensitivity and mechanotransduction during heart developmentNuclear lamin stiffness is a barrier to 3D migration, but softness can limit survival.Material control of stem cell differentiation: challenges in nano-characterization.Cross-linked matrix rigidity and soluble retinoids synergize in nuclear lamina regulation of stem cell differentiationFractal heterogeneity in minimal matrix models of scars modulates stiff-niche stem-cell responses via nuclear exit of a mechanorepressor.Engineering cell sensing and responses using a GPCR-coupled CRISPR-Cas system.Engineering Cell Sensing and Responses Using a GPCR-Coupled CRISPR-Cas SystemSIRPA-Inhibited, Marrow-Derived Macrophages Engorge, Accumulate, and Differentiate in Antibody-Targeted Regression of Solid Tumors
P50
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P50
description
Amerikaans onderzoeker
@nl
researcher ORCID ID = 0000-0002-1052-2537
@en
name
P. C. Dave P. Dingal
@ast
P. C. Dave P. Dingal
@en
P. C. Dave P. Dingal
@es
P. C. Dave P. Dingal
@nl
type
label
P. C. Dave P. Dingal
@ast
P. C. Dave P. Dingal
@en
P. C. Dave P. Dingal
@es
P. C. Dave P. Dingal
@nl
prefLabel
P. C. Dave P. Dingal
@ast
P. C. Dave P. Dingal
@en
P. C. Dave P. Dingal
@es
P. C. Dave P. Dingal
@nl
P106
P31
P496
0000-0002-1052-2537