about
Therapies for sarcopenia and regeneration of old skeletal muscles: more a case of old tissue architecture than old stem cellsNew horizons in the pathogenesis, diagnosis and management of sarcopeniaSelective modulation through the glucocorticoid receptor ameliorates muscle pathology in mdx miceImmunobiology and the future of myoblast transfer therapy.The different impact of a high fat diet on dystrophic mdx and control C57Bl/10 miceStriking denervation of neuromuscular junctions without lumbar motoneuron loss in geriatric mouse muscle.Reasons for the degeneration of ageing skeletal muscle: a central role for IGF-1 signalling.Problems and solutions in myoblast transfer therapy.Dystropathology increases energy expenditure and protein turnover in the mdx mouse model of duchenne muscular dystrophyEnhancing translation: guidelines for standard pre-clinical experiments in mdx miceInteractions between Skeletal Muscle Myoblasts and their Extracellular Matrix Revealed by a Serum Free Culture System.Quantification of ceroid and lipofuscin in skeletal muscle.MicroRNA expression patterns in post-natal mouse skeletal muscle development.Harnessing the therapeutic potential of myogenic stem cells.Strength at the extracellular matrix-muscle interface.Muscle-derived stem cells: implications for effective myoblast transfer therapy.Of bears, frogs, meat, mice and men: complexity of factors affecting skeletal muscle mass and fat.Duchenne muscular dystrophy: focus on pharmaceutical and nutritional interventions.The allure of stem cell therapy for muscular dystrophy.Towards developing standard operating procedures for pre-clinical testing in the mdx mouse model of Duchenne muscular dystrophyImplications of cross-talk between tumour necrosis factor and insulin-like growth factor-1 signalling in skeletal muscle.Two-tiered hypotheses for Duchenne muscular dystrophy.Modified patient stem cells as prelude to autologous treatment of muscular dystrophy.Oxidative stress as a therapeutic target during muscle wasting: considering the complex interactions.Levels of inflammation and oxidative stress, and a role for taurine in dystropathology of the Golden Retriever Muscular Dystrophy dog model for Duchenne Muscular Dystrophy.Imaging deep skeletal muscle structure using a high-sensitivity ultrathin side-viewing optical coherence tomography needle probe.Voluntary resistance wheel exercise from mid-life prevents sarcopenia and increases markers of mitochondrial function and autophagy in muscles of old male and female C57BL/6J mice.Optical coherence tomography can assess skeletal muscle tissue from mouse models of muscular dystrophy by parametric imaging of the attenuation coefficient.Oxidative stress and pathology in muscular dystrophies: focus on protein thiol oxidation and dysferlinopathies.Skeletal muscle degeneration and regeneration in mice and flies.Differential thiol oxidation of the signaling proteins Akt, PTEN or PP2A determines whether Akt phosphorylation is enhanced or inhibited by oxidative stress in C2C12 myotubes derived from skeletal muscle.Targeting macrophages rescues age-related immune deficiencies in C57BL/6J geriatric mice.Increasing taurine intake and taurine synthesis improves skeletal muscle function in the mdx mouse model for Duchenne muscular dystrophy.Innate inflammatory cells are not responsible for early death of donor myoblasts after myoblast transfer therapy.Taurine deficiency, synthesis and transport in the mdx mouse model for Duchenne Muscular Dystrophy.Extracellular matrix, growth factors, genetics: their influence on cell proliferation and myotube formation in primary cultures of adult mouse skeletal muscle.Silk fibroin scaffolds with muscle-like elasticity support in vitro differentiation of human skeletal muscle cells.Erratum to: Voluntary resistance wheel exercise from mid-life prevents sarcopenia and increases markers of mitochondrial function and autophagy in muscles of old male and female C57BL/6J mice.Use of pifithrin to inhibit p53-mediated signalling of TNF in dystrophic muscles of mdx mice.Protein thiol oxidation does not change in skeletal muscles of aging female mice.
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Miranda Grounds
@ast
Miranda Grounds
@en
Miranda Grounds
@es
Miranda Grounds
@nl
type
label
Miranda Grounds
@ast
Miranda Grounds
@en
Miranda Grounds
@es
Miranda Grounds
@nl
prefLabel
Miranda Grounds
@ast
Miranda Grounds
@en
Miranda Grounds
@es
Miranda Grounds
@nl
P106
P1153
7006703552
P31
P496
0000-0002-4530-9402