about
Connecting variability in global transcription rate to mitochondrial variabilityMitochondrial variability as a source of extrinsic cellular noiseNOV (CCN3) functions as a regulator of human hematopoietic stem or progenitor cellsReceptor and secreted targets of Wnt-1/beta-catenin signalling in mouse mammary epithelial cellsFunctional and molecular characterisation of mammary side population cellsErratum to: Functional and molecular characterisation of mammary side population cellsRetroviral vectors for establishing tetracycline-regulated gene expression in an otherwise recalcitrant cell lineIsoform-specific potentiation of stem and progenitor cell engraftment by AML1/RUNX1Interactions of GATA-2 with the promyelocytic leukemia zinc finger (PLZF) protein, its homologue FAZF, and the t(11;17)-generated PLZF-retinoic acid receptor alpha oncoproteinDNA repair is limiting for haematopoietic stem cells during ageingHaploinsufficiency of GATA-2 perturbs adult hematopoietic stem-cell homeostasisTranscriptional regulation of lineage commitment--a stochastic model of cell fate decisions.Coordinate gene regulation during hematopoiesis is related to genomic organizationComputational modeling of the hematopoietic erythroid-myeloid switch reveals insights into cooperativity, priming, and irreversibilityHigh GATA-2 expression inhibits human hematopoietic stem and progenitor cell function by effects on cell cycle.Directing oncogenic fusion genes into stem cells via an SCL enhancer.Single-cell entropy for accurate estimation of differentiation potency from a cell's transcriptomeMouse xenograft modeling of human adult acute lymphoblastic leukemia provides mechanistic insights into adult LIC biology.Forcing cells to change lineages.Generation of bivalent chromatin domains during cell fate decisionsGATA-2 and GATA-2/ER display opposing activities in the development and differentiation of blood progenitors.Transcription factor-mediated lineage switching reveals plasticity in primary committed progenitor cells.Modeling first-hit functions of the t(12;21) TEL-AML1 translocation in mice.A biophysical model for transcription factories.Plasticity revisited.Molecular complexities of stem cells.Early dynamic fate changes in haemogenic endothelium characterized at the single-cell level.Culture adaptation alters transcriptional hierarchies among single human embryonic stem cells reflecting altered patterns of differentiation.Role of the TEL-AML1 fusion gene in the molecular pathogenesis of childhood acute lymphoblastic leukaemia.GATA-2 mediated regulation of normal hematopoietic stem/progenitor cell function, myelodysplasia and myeloid leukemia.Developmental impact of leukemic fusion genes on stem cell fate.Single-Cell Analysis Identifies Distinct Stages of Human Endothelial-to-Hematopoietic Transition.Quiescent leukaemic cells account for minimal residual disease in childhood lymphoblastic leukaemiaInference, validation, and dynamic modeling of transcription networks in multipotent hematopoietic cells.The TEL-AML1 leukemia fusion gene dysregulates the TGF-beta pathway in early B lineage progenitor cells.Dynamic analysis of gene expression and genome-wide transcription factor binding during lineage specification of multipotent progenitors.Stem cell states, fates, and the rules of attraction.A network including TGFβ/Smad4, Gata2, and p57 regulates proliferation of mouse hematopoietic progenitor cells.Primed and ready: understanding lineage commitment through single cell analysis.Herpesviruses shape tumour microenvironment through exosomal transfer of viral microRNAs.
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P31
P496
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