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Podocyte-associated talin1 is critical for glomerular filtration barrier maintenance.Acute podocyte vascular endothelial growth factor (VEGF-A) knockdown disrupts alphaVbeta3 integrin signaling in the glomerulus.Renalase prevents AKI independent of amine oxidase activityRole of medullary progenitor cells in epithelial cell migration and proliferation.Podocyte-specific VEGF-a gain of function induces nodular glomerulosclerosis in eNOS null mice.TGF-beta receptor deletion in the renal collecting system exacerbates fibrosis.Hypertonicity-induced mitochondrial membrane permeability in renal medullary interstitial cells: protective role of osmolytes.Lack of integrin alpha1beta1 leads to severe glomerulosclerosis after glomerular injury.Loss of polycystin-1 inhibits Bicc1 expression during mouse development.Chemokine receptor Cxcr4 contributes to kidney fibrosis via multiple effectors.Semaphorin3a promotes advanced diabetic nephropathyComparison of amyloid deposition in human kidney biopsies as predictor of poor patient outcome.GM-CSF Promotes Macrophage Alternative Activation after Renal Ischemia/Reperfusion Injury.Isoforms of Spectrin and Ankyrin Reflect the Functional Topography of the Mouse Kidney.Human polycystin-2 transgene dose-dependently rescues ADPKD phenotypes in Pkd2 mutant mice.Three-Dimensional Morphology by Multiphoton Microscopy with Clearing in a Model of Cisplatin-Induced CKD.Essential Role of X-Box Binding Protein-1 during Endoplasmic Reticulum Stress in Podocytes.Apoptosis of the thick ascending limb results in acute kidney injury.NFATc1 identifies a population of proximal tubule cell progenitors.Placental insufficiency associated with loss of Cited1 causes renal medullary dysplasiaDistinct roles for basal and induced COX-2 in podocyte injuryAlloantibody and complement promote T cell-mediated cardiac allograft vasculopathy through noncanonical nuclear factor-κB signaling in endothelial cells.Met activation is required for early cytoprotection after ischemic kidney injury.Mineralocorticoid receptor phosphorylation regulates ligand binding and renal response to volume depletion and hyperkalemiaNephrotoxicity from chemotherapeutic agents: clinical manifestations, pathobiology, and prevention/therapy.Histones and Neutrophil Extracellular Traps Enhance Tubular Necrosis and Remote Organ Injury in Ischemic AKI.Aldosterone stimulates fibronectin synthesis in renal fibroblasts through mineralocorticoid receptor-dependent and independent mechanisms.Early B-cell factor 1 is an essential transcription factor for postnatal glomerular maturationEculizumab Therapy for Chronic Antibody-Mediated Injury in Kidney Transplant Recipients: A Pilot Randomized Controlled Trial.Proteinuria and glomerular injury associated with the anti-angiogenesis drug Vargatef™.Warfarin-related nephropathy in a patient with mild IgA nephropathy on dabigatran and aspirin.A Nodular Foreign Body Reaction in a Dialysis Patient Receiving Long-term Treatment With Lanthanum Carbonate.Rapamycin treatment dose-dependently improves the cystic kidney in a new ADPKD mouse model via the mTORC1 and cell-cycle-associated CDK1/cyclin axis.Loss of the podocyte glucocorticoid receptor exacerbates proteinuria after injury.Podocyte vascular endothelial growth factor (Vegf₁₆₄) overexpression causes severe nodular glomerulosclerosis in a mouse model of type 1 diabetes.Quantification and localization of M2 macrophages in human kidneys with acute tubular injury.Cytomegalovirus glomerulopathy and cytomegalovirus interstitial nephritis on sequential transplant kidney biopsies.Late-onset omeprazole-associated acute interstitial nephritis.Hypertonic stress and cell death. Focus on "Multiple cell death pathways are independently activated by lethal hypertonicity in renal epithelial cells".Importance of functional EGF receptors in recovery from acute nephrotoxic injury.
P50
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P50
description
researcher
@en
name
Gilbert W Moeckel
@en
Gilbert W Moeckel
@nl
type
label
Gilbert W Moeckel
@en
Gilbert W Moeckel
@nl
prefLabel
Gilbert W Moeckel
@en
Gilbert W Moeckel
@nl
P106
P108
P31
P496
0000-0002-6059-0837