Met activation is required for early cytoprotection after ischemic kidney injury. - Wikidata - wikimedia - TriplyDB

Met activation is required for early cytoprotection after ischemic kidney injury.

Met activation is required for early cytoprotection after ischemic kidney injury.

http://www.wikidata.org/entity/Q37524124

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Hepatocyte growth factor (Hgf) stimulates low density lipoprotein receptor-related protein (Lrp) 5/6 phosphorylation and promotes canonical Wnt signaling Suppressed autophagic response underlies augmentation of renal ischemia/reperfusion injury by type 2 diabetes.Chemokine receptor Cxcr4 contributes to kidney fibrosis via multiple effectors.Three-Dimensional Morphology by Multiphoton Microscopy with Clearing in a Model of Cisplatin-Induced CKD.Renoprotective approaches and strategies in acute kidney injury The emerging role of hepatocyte growth factor in renal diseases.Late intervention with the small molecule BB3 mitigates postischemic kidney injury.Biological roles of hepatocyte growth factor-Met signaling from genetically modified animals.Transcription Factor Trps1 Promotes Tubular Cell Proliferation after Ischemia-Reperfusion Injury through cAMP-Specific 3',5'-Cyclic Phosphodiesterase 4D and AKT.Protective effect of luteolin on skin ischemia-reperfusion injury through an AKT-dependent mechanism

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Met activation is required for early cytoprotection after ischemic kidney injury.

http://www.wikidata.org/entity/Q37524124

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

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scientific article published on 17 October 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Met activation is required for early cytoprotection after ischemic kidney injury.

@en

Met activation is required for early cytoprotection after ischemic kidney injury.

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type

label

Met activation is required for early cytoprotection after ischemic kidney injury.

@en

Met activation is required for early cytoprotection after ischemic kidney injury.

@nl

prefLabel

Met activation is required for early cytoprotection after ischemic kidney injury.

@en

Met activation is required for early cytoprotection after ischemic kidney injury.

@nl

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Journal of the American Society of Nephrology

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Met activation is required for early cytoprotection after ischemic kidney injury

@en

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Arnaud Marlier

http://www.w3.org/2001/XMLSchema#string

Carlos Hader

http://www.w3.org/2001/XMLSchema#string

Lloyd G Cantley

http://www.w3.org/2001/XMLSchema#string

Sherene Mason

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P2860

G1 cell cycle progression and the expression of G1 cyclins are regulated by PI3K/AKT/mTOR/p70S6K1 signaling in human ovarian cancer cells

The phosphoinositide 3-kinase pathway

TOR signaling in growth and metabolism

PDGF- and insulin-dependent pp70S6k activation mediated by phosphatidylinositol-3-OH kinase

Disparate mechanisms for hypoxic cell injury in different nephron segments. Studies in the isolated perfused rat kidney

Coupling of Gab1 to c-Met, Grb2, and Shp2 mediates biological responses

Evidence that fibroblasts derive from epithelium during tissue fibrosis

Growth factor-mediated phosphorylation of proapoptotic BAD reduces tubule cell death in vitro and in vivo.

Met and the epidermal growth factor receptor act cooperatively to regulate final nephron number and maintain collecting duct morphology

Phosphatidylinositol 3-kinase signals activation of p70 S6 kinase in situ through site-specific p70 phosphorylation

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329-337

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scholarly article

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10.1681/ASN.2013050473

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2

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25

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Gilbert W Moeckel

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2013-10-17T00:00:00Z

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24136921

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3904569

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